Document Detail


Neutrophil IL-10 suppresses peritoneal inflammatory monocytes during polymicrobial sepsis.
MedLine Citation:
PMID:  21106642     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Septic peritonitis remains a major cause of death. Neutrophils and inflammatory monocytes are principal components of the innate immune system and are essential for defense against a range of microbial pathogens. Their role and interaction in polymicrobial sepsis have not been defined clearly. Using a murine model of CLP to induce moderate sepsis, we found that neutrophil depletion did not alter survival, whereas depletion of neutrophils and inflammatory monocytes markedly reduced survival. After neutrophil depletion, inflammatory monocytes had greater phagocytic capacity and oxidative burst, and increased expression of costimulatory molecules, TNF, and iNOS. Notably, peritoneal neutrophils produced IL-10 following CLP. Adoptive i.p. transfer of WT but not IL-10(-/-) neutrophils into septic mice reduced monocyte expression of TNF. In vitro experiments confirmed that monocyte suppression was mediated by neutrophil-derived IL-10. Thus, during septic peritonitis, neutrophils suppress peritoneal inflammatory monocytes through IL-10 and are dispensable for survival.
Authors:
Lee M Ocuin; Zubin M Bamboat; Vinod P Balachandran; Michael J Cavnar; Hebroon Obaid; George Plitas; Ronald P DeMatteo
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2010-11-24
Journal Detail:
Title:  Journal of leukocyte biology     Volume:  89     ISSN:  1938-3673     ISO Abbreviation:  J. Leukoc. Biol.     Publication Date:  2011 Mar 
Date Detail:
Created Date:  2011-03-01     Completed Date:  2011-04-20     Revised Date:  2012-03-01    
Medline Journal Info:
Nlm Unique ID:  8405628     Medline TA:  J Leukoc Biol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  423-32     Citation Subset:  IM    
Affiliation:
Hepatopancreatobiliary Service, Memorial Sloan-Kettering Cancer Center, New York, New York, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Inflammation / complications,  immunology,  pathology
Interleukin-10 / immunology*
Male
Mice
Mice, Inbred C57BL
Monocytes / enzymology,  pathology*
Neutrophils / enzymology,  immunology*
Nitric Oxide Synthase Type II / metabolism
Peritonitis / complications*,  immunology*,  pathology
Sepsis / complications,  immunology*,  microbiology*,  pathology
Survival Analysis
Tumor Necrosis Factor-alpha / metabolism
Grant Support
ID/Acronym/Agency:
AI70658/AI/NIAID NIH HHS; DK068346/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Tumor Necrosis Factor-alpha; 130068-27-8/Interleukin-10; EC 1.14.13.39/Nitric Oxide Synthase Type II

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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