Document Detail


Neurotensin-induced hypothermia improves neurologic outcome after hypoxic-ischemia.
MedLine Citation:
PMID:  15090966     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: External cooling is commonly used to force induction of mild hypothermia but requires equipment, has a slow onset of action, and must be prolonged to provide permanent neurologic benefits after hypoxic-ischemia. It is unknown whether the method for inducing mild hypothermia affects neurologic outcome after near-drowning. The objective of the study was to induce mild hypothermia with neurotensin analog NT77 or external cooling in a rat model of near-drowning. We hypothesize that NT77 would be more effective for improving neurologic outcome than external cooling of the same duration. DESIGN: Rats were randomized to a normothermic control, neurotensin-induced hypothermia, brief external cooling, or prolonged external cooling group after asphyxial cardiac arrest. SETTING: Laboratory investigation. SUBJECTS: Forty-eight rats. INTERVENTIONS: Mild hypothermia was induced by external cooling for 4 hrs (brief external cooling) or 24 hrs (prolonged external cooling) or by neurotensin-induced hypothermia administration 30 mins after asphyxial cardiac arrest in rats. MEASUREMENTS: Outcome was assessed by a neurologic deficit score, the Morris water maze, and CA1 hippocampus histology 15 days after resuscitation. MAIN RESULTS: Neurologic deficit score at 72 hrs after asphyxial cardiac arrest was lower with neurotensin-induced hypothermia (score, 0) and prolonged external cooling (score, 0) vs. normothermic control (score, 20) and brief external cooling (score, 18; p <.05). Latency time in the Morris water maze 15 days after asphyxial cardiac arrest was decreased with neurotensin-induced hypothermia (14+/-11 secs) and prolonged external cooling (18+/-9 secs) vs. normothermic control (74+/-17 secs) and brief external cooling (78+/-18 secs, p <.05). There was less ischemic neuronal damage with neurotensin-induced hypothermia (28+/-24%) and prolonged external cooling (21+/-14%) vs. normothermic control (61+/-32%) and brief external cooling (51+/-32%). CONCLUSIONS: Neurotensin-induced hypothermia improved neurologic outcome after asphyxial cardiac arrest in rats vs. brief external cooling but was comparable to prolonged external cooling.
Authors:
Laurence M Katz; Amanda Young; Jonathan E Frank; Yuanfan Wang; Kyunam Park
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Publication Detail:
Type:  Evaluation Studies; Journal Article    
Journal Detail:
Title:  Critical care medicine     Volume:  32     ISSN:  0090-3493     ISO Abbreviation:  Crit. Care Med.     Publication Date:  2004 Mar 
Date Detail:
Created Date:  2004-04-19     Completed Date:  2004-04-30     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0355501     Medline TA:  Crit Care Med     Country:  United States    
Other Details:
Languages:  eng     Pagination:  806-10     Citation Subset:  AIM; IM    
Affiliation:
University of North Carolina School of Medicine, Chapel Hill, NC, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Asphyxia / therapy
Disease Models, Animal
Heart Arrest / therapy
Hypothermia, Induced / methods*
Hypoxia, Brain / prevention & control*
Near Drowning / therapy*
Neurotensin / analogs & derivatives*,  therapeutic use*
Random Allocation
Rats
Statistics, Nonparametric
Chemical
Reg. No./Substance:
39379-15-2/Neurotensin
Comments/Corrections
Comment In:
Crit Care Med. 2004 Mar;32(3):897-9   [PMID:  15090990 ]

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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