Document Detail


Neuronal nitric oxide synthase and splanchnic blood flow in anaesthetized rats.
MedLine Citation:
PMID:  15743385     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
AIMS: To evaluate to what extent the neuronal form of constitutive nitric oxide synthase (nNOS) contributes to the blood perfusion of splanchnic organs, including the islets of Langerhans. METHODS: The nNOS inhibitor 7-nitroindazole (300 mg kg(-1) i.p.) was administered to anaesthetized Sprague-Dawley rats, some of which were pre-treated with the ganglionic blocker hexamethonium (20 mg kg(-1) i.v.) The blood perfusion of the splanchnic organs, including the pancreatic islets was then measured with a microsphere technique. RESULTS: Nitroindazole decreased total pancreatic, duodenal and renal blood flow, whereas pancreatic islet, colonic and adrenal blood flows were unchanged. A slight increase in mean arterial blood pressure was seen after nitroindazole treatment. Nitroindazole did not affect blood glucose or serum insulin concentrations. In separate experiments, hexamethonium affected none of the studied blood flow values, suggesting that the effects of nNOS-inhibition were not mediated from the nervous system. CONCLUSION: Nitric oxide derived from the activity of nNOS contributes to the blood perfusion in the upper portions of the gastrointestinal tract, viz. the parts supplied by the cranial mesenteric artery, and the kidneys, whilst no effects are seen on colonic or adrenal blood flow. Pancreatic islet blood flow was unaffected by nNOS inhibition, thereby suggesting that NO derived from the other isoforms of NOS maintains the high basal islet blood perfusion.
Authors:
L Jansson; P-O Carlsson; B Bodin; A Andersson; O Källskog
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Acta physiologica Scandinavica     Volume:  183     ISSN:  0001-6772     ISO Abbreviation:  Acta Physiol. Scand.     Publication Date:  2005 Mar 
Date Detail:
Created Date:  2005-03-03     Completed Date:  2005-05-03     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0370362     Medline TA:  Acta Physiol Scand     Country:  England    
Other Details:
Languages:  eng     Pagination:  257-62     Citation Subset:  IM    
Affiliation:
Department of Medical Cell Biology, Biomedical Center, Uppsala University, SE 751 23 Uppsala, Sweden.
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MeSH Terms
Descriptor/Qualifier:
Anesthesia, General
Animals
Blood Glucose / metabolism
Blood Pressure / drug effects
Enzyme Inhibitors / pharmacology
Gastrointestinal Tract / blood supply
Hexamethonium / pharmacology
Indazoles / pharmacology
Insulin / blood
Islets of Langerhans / blood supply
Male
Nerve Tissue Proteins / antagonists & inhibitors,  physiology*
Nitric Oxide Synthase / antagonists & inhibitors,  physiology*
Nitric Oxide Synthase Type I
Rats
Rats, Sprague-Dawley
Renal Circulation / drug effects,  physiology
Splanchnic Circulation / drug effects,  physiology*
Chemical
Reg. No./Substance:
0/Blood Glucose; 0/Enzyme Inhibitors; 0/Indazoles; 0/Nerve Tissue Proteins; 11061-68-0/Insulin; 2942-42-9/7-nitroindazole; 60-26-4/Hexamethonium; EC 1.14.13.39/Nitric Oxide Synthase; EC 1.14.13.39/Nitric Oxide Synthase Type I; EC 1.14.13.39/Nos1 protein, rat

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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