| Neurohumoral pathways in heart failure with preserved systolic function. | |
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MedLine Citation:
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PMID: 16115515 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Other than natriuretic peptides, neurohumoral markers have not been studied adequately in heart failure with preserved systolic function. There is little evidence of chronic activation of the renin-angiotensin-aldosterone and sympathetic nervous systems, as in heart failure with reduced systolic function. These and other neurohumoral pathways may, however, become excessively stimulated by exercise and during episodes of acute decompensation. Blood natriuretic peptide concentrations are chronically elevated in patients with heart failure and preserved systolic function but not to the same extent as in patients with reduced systolic function. The role of neurohumoral antagonists in the treatment of heart failure and preserved systolic function has yet to be fully investigated. |
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Authors:
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Karen Hogg; John McMurray |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Progress in cardiovascular diseases Volume: 47 ISSN: 0033-0620 ISO Abbreviation: Prog Cardiovasc Dis Publication Date: 2005 May-Jun |
Date Detail:
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Created Date: 2005-08-23 Completed Date: 2005-10-06 Revised Date: 2007-11-15 |
Medline Journal Info:
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Nlm Unique ID: 0376442 Medline TA: Prog Cardiovasc Dis Country: United States |
Other Details:
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Languages: eng Pagination: 357-66 Citation Subset: AIM; IM |
Affiliation:
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Department of Cardiology, Western Infirmary, Glasgow, Scotland. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Heart Failure
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physiopathology* Humans Natriuretic Peptides / physiology* Neurotransmitter Agents / physiology* Sympathetic Nervous System / physiopathology Systole / physiology* Ventricular Function / physiology* |
| Chemical | |
Reg. No./Substance:
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0/Natriuretic Peptides; 0/Neurotransmitter Agents |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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