Document Detail


Neurofunctional effects of developmental alcohol exposure in alcohol-preferring and alcohol-nonpreferring rats.
MedLine Citation:
PMID:  11331149     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The neurofunctional effects of developmental alcohol exposure (3% v/v solution from day 15 of gestation to day 7 after parturition) have been investigated in Sardinian alcohol-preferring (sP) and alcohol-nonpreferring (sNP) rat lines, selectively bred for opposite alcohol preference and consumption. Alcohol exposure significantly decreased the rate of ultrasonic emission in sP male pups; whereas, it did not affect this indicator of emotional reactivity in sNP animals. Perinatal alcohol intake did not influence either learning of an active avoidance task or hippocampal long-term potentiation in both offspring lines. Significant differences in time spent exploring novel objects were observed between control sP and sNP rats subjected to the novel exploration object test. Alcohol exposed sP rats, but not alcohol exposed sNP rats, apparently lost the capacity to discriminate between the novel and the familiar object, even though this difference is difficult to interpret because of the large differences in the respective responses to the novel objects. Neurochemical experiments have shown that basal levels of dopamine (DA) and homovanillic acid (HVA) were significantly higher in the nucleus accumbens (NAC) of sP rats with respect to sNP animals. Perinatal alcohol did not affect basal DA and HVA concentrations or amphetamine-induced DA increase and HVA decrease in the NAC of either sP or sNP offspring. These results suggest that subtle behavioral alterations induced by developmental exposure to low doses of alcohol, which do not cause malformations and/or overt neurotoxicity, may be associated with genetic factors, although not necessarily those responsible for differences in alcohol preference.
Authors:
M Tattoli; R Cagiano; S Gaetani; V Ghiglieri; A Giustino; G Mereu; L Trabace; V Cuomo
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology     Volume:  24     ISSN:  0893-133X     ISO Abbreviation:  Neuropsychopharmacology     Publication Date:  2001 Jun 
Date Detail:
Created Date:  2001-05-01     Completed Date:  2001-07-19     Revised Date:  2011-05-18    
Medline Journal Info:
Nlm Unique ID:  8904907     Medline TA:  Neuropsychopharmacology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  691-705     Citation Subset:  IM    
Affiliation:
Department of Pharmacology and Human Physiology, University of Bari, Bari, Italy.
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MeSH Terms
Descriptor/Qualifier:
Alcohol-Induced Disorders, Nervous System / congenital*,  physiopathology*
Alcoholism / physiopathology*
Amphetamine / pharmacology
Animals
Avoidance Learning / drug effects,  physiology
Brain / drug effects*,  embryology,  growth & development
Dopamine / metabolism
Ethanol / blood
Exploratory Behavior / drug effects,  physiology
Female
Fetal Alcohol Syndrome / physiopathology*
Food Preferences / physiology*
Hippocampus / drug effects,  physiology
Long-Term Potentiation / drug effects,  physiology
Male
Motor Activity / drug effects,  physiology
Nucleus Accumbens / drug effects,  metabolism
Pregnancy
Prenatal Exposure Delayed Effects*
Rats
Rats, Wistar
Vocalization, Animal / drug effects,  physiology
Chemical
Reg. No./Substance:
300-62-9/Amphetamine; 64-17-5/Ethanol

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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