Document Detail


Neuroblasts: a model for the asymmetric division of stem cells.
MedLine Citation:
PMID:  9009846     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The ability of stem cells to self-renew has hung been attributed to an asymmetry in division that generates one daughter cell identical to the mother and another cell committed to differentiation. Recent studies on neuroblasts, a group of neural stem cells responsible for generating various neurons and glial cells in the central nervous system, have revealed exciting mechanisms that underlie self-renewing asymmetric division. Several important localized cell fate determinants have been characterized, and their segregation mechanism has been explored in the context of cytoskeletal organization, cell-cycle type progression, cytokinesis and mitotic orientation. These findings are illuminating in understanding the general mechanism of stem cell division.
Authors:
H Lin; T Schagat
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Trends in genetics : TIG     Volume:  13     ISSN:  0168-9525     ISO Abbreviation:  Trends Genet.     Publication Date:  1997 Jan 
Date Detail:
Created Date:  1997-02-27     Completed Date:  1997-02-27     Revised Date:  2008-10-16    
Medline Journal Info:
Nlm Unique ID:  8507085     Medline TA:  Trends Genet     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  33-9     Citation Subset:  IM    
Affiliation:
Department of Cell Biology, Duke University Medical School, Durham, NC 27710, USA. halfan_lin@cellbio.duke.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Differentiation
Cell Division
Drosophila / embryology,  physiology
Drosophila Proteins*
Gene Expression Regulation, Developmental
Juvenile Hormones / physiology
Mammals / physiology
Membrane Proteins / physiology
Models, Biological
Nerve Tissue Proteins / physiology
Neurons / cytology*,  physiology*
Nuclear Proteins / physiology
Receptors, Notch
Signal Transduction
Stem Cells / cytology,  physiology*
Transcription Factors*
Grant Support
ID/Acronym/Agency:
R01HD33760-01/HD/NICHD NIH HHS
Chemical
Reg. No./Substance:
0/Drosophila Proteins; 0/Juvenile Hormones; 0/Membrane Proteins; 0/Nerve Tissue Proteins; 0/Nuclear Proteins; 0/Receptors, Notch; 0/Transcription Factors; 0/notch protein, Drosophila; 0/numb protein, Drosophila; 0/pros protein, Drosophila

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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