Document Detail


Natural and azido fatty acids inhibit phosphate transport and activate fatty acid anion uniport mediated by the mitochondrial phosphate carrier.
MedLine Citation:
PMID:  11085992     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The electroneutral P(i) uptake via the phosphate carrier (PIC) in rat liver and heart mitochondria is inhibited by fatty acids (FAs), by 12-(4-azido-2-nitrophenylamino)dodecanoic acid (AzDA) and heptylbenzoic acid ( approximately 1 microm doses) and by lauric, palmitic, or 12-azidododecanoic acids ( approximately 0.1 mm doses). In turn, reconstituted E. coli-expressed yeast PIC mediated anionic FA uniport with a similar pattern leading to FA cycling and H(+) uniport. The kinetics of P(i)/P(i) exchange on recombinant PIC in the presence of AzDA better corresponded to a competitive inhibition mechanism. Methanephosphonate was identified as a new PIC substrate. Decanephosphonate, butanephosphonate, 4-nitrophenylphosphate, and other P(i) analogs were not translocated and did not inhibit P(i) transport. However, methylenediphosphonate and iminodi(methylenephosphonate) inhibited both electroneutral P(i) uptake and FA cycling via PIC. AzDA analog 16-(4-azido-2-nitrophenylamino)-[(3)H(4)]-hexadecanoic acid ((3)H-AzHA) bound upon photoactivation to several mitochondrial proteins, including the 30- and 34-kDa bands. The latter was ascribed to PIC due to its specific elution pattern on Blue Sepharose and Affi-Gel. (3)H-AzHA photolabeling of recombinant PIC was prevented by methanephosphonate and diphosphonates and after premodification with 4-azido-2-nitrophenylphosphate. Hence, the demonstrated PIC interaction with monovalent long-chain FA anions, but with divalent phosphonates of short chain only, indicates a pattern distinct from that valid for the mitochondrial uncoupling protein-1.
Authors:
H Engstová; M Zácková; M Růzicka; A Meinhardt; J Hanus; R Krämer; P Jezek
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2000-11-20
Journal Detail:
Title:  The Journal of biological chemistry     Volume:  276     ISSN:  0021-9258     ISO Abbreviation:  J. Biol. Chem.     Publication Date:  2001 Feb 
Date Detail:
Created Date:  2001-05-23     Completed Date:  2001-06-28     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  2985121R     Medline TA:  J Biol Chem     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4683-91     Citation Subset:  IM    
Affiliation:
Institute of Physiology, Department of Membrane Transport Biophysics, Academy of Sciences of the Czech Republic, Prague CZ 14220, Czech Republic.
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MeSH Terms
Descriptor/Qualifier:
Affinity Labels / pharmacology
Animals
Biological Transport, Active
Carrier Proteins / antagonists & inhibitors*,  metabolism*
Diphosphonates / pharmacology
Dose-Response Relationship, Drug
Fatty Acids / metabolism,  pharmacology*
Ion Transport
Kinetics
Lauric Acids / pharmacology*
Mitochondria / drug effects,  metabolism
Palmitic Acids / pharmacology
Phosphate-Binding Proteins
Phosphates / metabolism*
Rats
Rats, Wistar
Chemical
Reg. No./Substance:
0/12-(4-azido-2-nitrophenylamino)dodecanoic acid; 0/16-(4-azido-2-nitrophenylamino)hexadecanoic acid; 0/Affinity Labels; 0/Carrier Proteins; 0/Diphosphonates; 0/Fatty Acids; 0/Lauric Acids; 0/Palmitic Acids; 0/Phosphate-Binding Proteins; 0/Phosphates

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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