Document Detail


Nandrolone and resistance training induce heart remodeling: Role of fetal genes and implications for cardiac pathophysiology.
MedLine Citation:
PMID:  21889516     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
AIMS: This study was conducted to assess the isolated and combined effects of nandrolone and resistance training on cardiac morphology, function, and mRNA expression of pathological cardiac hypertrophy markers. MAIN METHODS: Wistar rats were randomly divided into four groups and submitted to 6weeks of treatment with nandrolone and/or resistance training. Cardiac parameters were determined by echocardiography. The heart was analyzed for collagen infiltration. Real-time RT-PCR was used to assess the pathological cardiac hypertrophy markers. KEY FINDINGS: Both resistance training and nandrolone induced cardiac hypertrophy. Nandrolone increased the cardiac collagen content, and reduced the cardiac index in non-trained and trained groups, when compared with the respective vehicle-treated groups. Nandrolone reduced the ratio of maximum early to late transmitral flow velocity in non-trained and trained groups, when compared with the respective vehicle-treated groups. Nandrolone reduced the alpha-myosin heavy chain gene expression in both non-trained and trained groups, when compared with the respective vehicle-treated groups. Training reduced the beta-myosin heavy chain gene expression in the groups treated with vehicle and nandrolone. Only the association between training and nandrolone increased the expression of the skeletal alpha-actin gene and atrial natriuretic peptide in the left ventricle. SIGNIFICANCE: This study indicated that nandrolone, whether associated with resistance training or not, induces cardiac hypertrophy, which is associated with enhanced collagen content, re-expression of fetal genes the in left ventricle, and impaired diastolic and systolic function.
Authors:
Ana Paula Tanno; Vander José das Neves; Kaleizu Teodoro Rosa; Tatiana Sousa Cunha; Fernanda Cristina Linarello Giordano; Caroline Morini Calil; Vinicius Guzzoni; Tiago Fernandes; Edilamar Menezes de Oliveira; Pedro Duarte Novaes; Maria Cláudia Irigoyen; Maria José Costa Sampaio Moura; Fernanda Klein Marcondes
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Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-8-17
Journal Detail:
Title:  Life sciences     Volume:  -     ISSN:  1879-0631     ISO Abbreviation:  -     Publication Date:  2011 Aug 
Date Detail:
Created Date:  2011-9-5     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0375521     Medline TA:  Life Sci     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Copyright Information:
Copyright © 2011. Published by Elsevier Inc.
Affiliation:
Department of Physiological Sciences, Piracicaba Dental School, University of Campinas, Piracicaba, SP, Brazil.
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