Document Detail


NOD2 mediates inflammatory responses of primary murine glia to Streptococcus pneumoniae.
MedLine Citation:
PMID:  20091781     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
It is now widely accepted that resident central nervous system (CNS) cells such as microglia and astrocytes initiate and/or augment inflammation following trauma or infection. However, the mechanisms by which glial cells perceive microbial challenges are only now becoming apparent. We have recently demonstrated that microglia and astrocytes constitutively express nucleotide-binding oligomerization domain-2 (NOD2), a member of the novel nucleotide-binding domain leucine-rich repeat region-containing family of proteins (NLR) that functions as an intracellular receptor for a minimal motif present in all bacterial peptidoglycans. Furthermore, we have shown that this NLR is essential for glial responses to gram-negative pathogens and in vivo CNS inflammation elicited by these organisms. In the present study, we have established that intact Streptococcus pneumoniae, the major causative agent for gram-positive bacterial meningitis in adults, is a potent stimulus for the activation of the pivotal inflammatory transcription factor NF-kB and production of inflammatory cytokines in primary murine microglia and astrocytes. We demonstrate that NOD2 is essential for the maximal responses of these cells to intact S. pneumoniae but not cellular lysates. Finally, we have shown that this cytosolic pattern recognition receptor is required for the elevated inflammatory mediator levels, astrogliosis, and demyelination, following in vivo administration of this gram-positive CNS pathogen. As such, we suggest that NOD2 plays a critical role in the establishment of the lethal inflammation associated with streptococcal meningitis.
Authors:
Xinjie Liu; Vinita S Chauhan; Amy B Young; Ian Marriott
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Glia     Volume:  58     ISSN:  1098-1136     ISO Abbreviation:  Glia     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-04-08     Completed Date:  2010-06-29     Revised Date:  2011-09-26    
Medline Journal Info:
Nlm Unique ID:  8806785     Medline TA:  Glia     Country:  United States    
Other Details:
Languages:  eng     Pagination:  839-47     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, Qilu Hospital, Shandong University, Jinan, Shandong, People's Republic of China.
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MeSH Terms
Descriptor/Qualifier:
Animals
Astrocytes / immunology,  metabolism
Cells, Cultured
Cytokines / metabolism
Encephalitis / immunology*,  microbiology*,  physiopathology
Gliosis / immunology*,  microbiology*,  physiopathology
Inflammation Mediators / metabolism
Meningitis, Bacterial / immunology,  metabolism,  physiopathology
Mice
Mice, Knockout
Microglia / immunology,  metabolism
NF-kappa B / metabolism
Nod2 Signaling Adaptor Protein / metabolism*
Streptococcal Infections / immunology*,  physiopathology
Grant Support
ID/Acronym/Agency:
NS050325/NS/NINDS NIH HHS; NS057434/NS/NINDS NIH HHS; R01 NS050325-04/NS/NINDS NIH HHS; R03 NS057434-02S1/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Card15 protein, mouse; 0/Cytokines; 0/Inflammation Mediators; 0/NF-kappa B; 0/Nod2 Signaling Adaptor Protein
Comments/Corrections

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