Document Detail


NMDA potentiation by visible light in the presence of a fluorescent neurosteroid analogue.
MedLine Citation:
PMID:  19403611     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
N-Methyl-D-aspartate (NMDA) receptors are widely studied because of their importance in synaptic plasticity and excitotoxic cell death. Here we report a novel method of potentiating NMDA receptors with fluorescence excited by blue (480 nm) light. In the presence of 300 nM of a (7-nitro-2,1,3-benzoxadiazol-4-yl) amino (NBD)-tagged neuroactive steroid carrier C2-NBD-(3alpha,5alpha)-3-hydroxypregnan-20-one (C2-NBD 3alpha5alphaP), responses of cultured hippocampal neurons to 10 microM NMDA were potentiated to 219.2 +/- 9.2% of the baseline response (100%) by a 30 s exposure to 480 nm light. The potentiation decayed back to baseline with a time constant of 80.6 s. Responses to 1 microM and 100 microM NMDA were potentiated to 147.9 +/- 9.6% and 174.1 +/- 15.6% of baseline, respectively, suggesting that visible-light potentiation is relatively insensitive to NMDA concentration. Peak autaptic NMDA responses were potentiated to 178.9 +/- 22.4% of baseline. Similar potentiation was seen with 10 microM NBD-lysine, suggesting that visible-light potentiation is not a steroid effect. Potentiation was also seen with a steroid analogue in which the NBD was replaced with fluorescein, suggesting that NBD is not the only fluorophore capable of supporting visible-light potentiation. UV light and redox potentiation of NMDA receptors largely occluded subsequent blue light potentiation (127.7 +/- 7.4% and 120.2 +/- 6.2% of baseline, respectively). The NR1a(C744A,C798A) mutant that is insensitive to redox and UV potentiation was also largely unaffected by visible-light potentiation (135.0 +/- 10.0% of baseline). Finally, we found that the singlet oxygen scavenger furfuryl alcohol decreased visible-light potentiation. Collectively, these data suggest that visible-light potentiation of NMDA receptors by fluorescence excitation shares mechanisms with UV and redox potentiation and may involve singlet oxygen production.
Authors:
Lawrence N Eisenman; Hong-Jin Shu; Cunde Wang; Elias Aizenman; Douglas F Covey; Charles F Zorumski; Steven Mennerick
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2009-04-29
Journal Detail:
Title:  The Journal of physiology     Volume:  587     ISSN:  1469-7793     ISO Abbreviation:  J. Physiol. (Lond.)     Publication Date:  2009 Jun 
Date Detail:
Created Date:  2009-06-15     Completed Date:  2009-09-21     Revised Date:  2010-09-27    
Medline Journal Info:
Nlm Unique ID:  0266262     Medline TA:  J Physiol     Country:  England    
Other Details:
Languages:  eng     Pagination:  2937-47     Citation Subset:  IM    
Affiliation:
Department of Neurology, Washington University School of Medicine, 660 S. Euclid Ave, Box 8111, St Louis, MO 63110, USA. leisenman@wustl.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cells, Cultured
Electrophysiology
Fluorescent Dyes*
Free Radical Scavengers / pharmacology
Furans / pharmacology
Hippocampus / chemistry,  physiology
Light*
Oxadiazoles*
Oxidation-Reduction
Patch-Clamp Techniques
Pregnanolone / analogs & derivatives*
Rats
Rats, Sprague-Dawley
Reactive Oxygen Species / metabolism
Receptors, GABA-A / drug effects
Receptors, N-Methyl-D-Aspartate / drug effects,  physiology*,  radiation effects*
Ultraviolet Rays
Grant Support
ID/Acronym/Agency:
AA12952/AA/NIAAA NIH HHS; GM 47969/GM/NIGMS NIH HHS; MH77791/MH/NIMH NIH HHS; MH78823/MH/NIMH NIH HHS; NS44041/NS/NINDS NIH HHS; NS54174/NS/NINDS NIH HHS; P30NS057105/NS/NINDS NIH HHS
Chemical
Reg. No./Substance:
0/Fluorescent Dyes; 0/Free Radical Scavengers; 0/Furans; 0/Oxadiazoles; 0/Reactive Oxygen Species; 0/Receptors, GABA-A; 0/Receptors, N-Methyl-D-Aspartate; 128-20-1/Pregnanolone; 98-00-0/furfuryl alcohol
Comments/Corrections

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