Document Detail


NK cell deficiency predisposes to viral-induced Th2-type allergic inflammation via epithelial-derived IL-25.
MedLine Citation:
PMID:  20855881     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Severe respiratory syncytial virus (RSV) infection has long been associated with an increased risk for the development of childhood asthma and exacerbations of this disorder. Despite much research into the induction of Th2 responses by allergens and helminths, the factors associated with viral infection that predispose to Th2-regulated asthma remain unknown. Recently, clinical studies have shown reduced numbers of NK cells in infants suffering from a severe RSV infection. Here we demonstrate that NK cell deficiency during primary RSV infection of BALB/c mice results in the suppression of IFN-γ production and the development of an RSV-specific Th2 response and subsequent allergic lung disease. The outgrowth of the Th2 responses was dependent on airway epithelial cell-derived IL-25, which induced the upregulation of the notch ligand Jagged1 on dendritic cells. This study identifies a novel pathway underlying viral-driven Th2 responses that may have functional relevance to viral-associated asthma.
Authors:
Gerard E Kaiko; Simon Phipps; Pornpimon Angkasekwinai; Chen Dong; Paul S Foster
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-09-20
Journal Detail:
Title:  Journal of immunology (Baltimore, Md. : 1950)     Volume:  185     ISSN:  1550-6606     ISO Abbreviation:  J. Immunol.     Publication Date:  2010 Oct 
Date Detail:
Created Date:  2010-10-06     Completed Date:  2010-11-04     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  2985117R     Medline TA:  J Immunol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  4681-90     Citation Subset:  AIM; IM    
Affiliation:
Centre for Asthma and Respiratory Diseases, School of Biomedical Sciences and Pharmacy, Faculty of Health, University of Newcastle, Newcastle, Australia.
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MeSH Terms
Descriptor/Qualifier:
Animals
Asthma / immunology*,  metabolism,  virology
Bronchoalveolar Lavage
Calcium-Binding Proteins / biosynthesis,  immunology
Cell Separation
Dendritic Cells / immunology,  metabolism
Enzyme-Linked Immunosorbent Assay
Epithelial Cells / immunology,  metabolism
Flow Cytometry
Hypersensitivity / immunology,  metabolism,  virology
Immunohistochemistry
Inflammation / immunology,  metabolism,  virology
Intercellular Signaling Peptides and Proteins / biosynthesis,  immunology
Interleukins / biosynthesis,  immunology*
Killer Cells, Natural / immunology*,  metabolism
Male
Membrane Proteins / biosynthesis,  immunology
Mice
Mice, Inbred BALB C
Respiratory Mucosa / immunology,  metabolism
Respiratory Syncytial Virus Infections / immunology*,  metabolism
Reverse Transcriptase Polymerase Chain Reaction
Th2 Cells / immunology*,  metabolism
Chemical
Reg. No./Substance:
0/Calcium-Binding Proteins; 0/D17Wsu104e protein, mouse; 0/Intercellular Signaling Peptides and Proteins; 0/Interleukins; 0/Membrane Proteins; 134324-36-0/Serrate proteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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