Document Detail


NAD(P)H fluorescence transients after synaptic activity in brain slices: predominant role of mitochondrial function.
MedLine Citation:
PMID:  16538234     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Excitatory stimulation in hippocampal slices results in biphasic NAD(P)H fluorescence transients. Previous studies using differing stimulus protocols agreed that the oxidation phase is a consequence of mitochondrial metabolism, but the reduction phase has been attributed to (1) mitochondrial nicotinamide adenine dinucleotide (NADH) generation or (2) astrocytic glycolysis triggered by glutamate uptake. In an attempt to reconcile these two views, the present study examined NAD(P)H signals evoked by a wide range of stimulus durations (40 ms to 20 secs). A combination of ionotropic glutamate receptor (iGluR) antagonists (6-cyano-7-nitroquinoxaline-2,3-dione (CNQX), 2-amino-5-phosphonopentanoic acid (APV)) virtually abolished responses to brief stimuli (40 to 200 ms, 50 Hz), but a significant fraction of the signal elicited by extended stimulation (20 secs, 32 Hz) was resistant to CNQX/APV. Glycolysis was inhibited by removal of glucose and addition of 2-deoxyglucose (2DG) (10 mmol/L) or iodoacetic acid (IAA, 1 mmol/L). Pyruvate was provided as an alternative substrate for oxidative phosphorylation and the A1 receptor antagonist 1,3-Dipropyl-8-cyclopentylxanthine (DPCPX) included to prevent decreases in synaptic efficacy. If sufficient pyruvate was supplied, responses to brief and extended stimuli were unaffected by glycolytic inhibition and not significantly reduced by an inhibitor of glucose uptake (3-O-methyl glucose, 3 mmol/L). When timed to arrive at the peak of overshoots generated by extended synaptic stimulation, brief pyruvate applications (10 mmol/L, 2 mins) had little effect on evoked NAD(P)H increases. Flavoprotein autofluorescence transients after extended stimuli matched (with inverted sign) NAD(P)H responses. Responses to extended stimuli were not reduced by a nonselective inhibitor of glutamate uptake DL-Threo-beta-benzyloxyaspartic acid (TBOA). These results suggest that NAD(P)H transients report mitochondrial dynamics, rather than recruitment of glycolytic metabolism, over a wide range of stimulus intensities.
Authors:
Angela M Brennan; John A Connor; C William Shuttleworth
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural     Date:  2006-03-15
Journal Detail:
Title:  Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism     Volume:  26     ISSN:  0271-678X     ISO Abbreviation:  J. Cereb. Blood Flow Metab.     Publication Date:  2006 Nov 
Date Detail:
Created Date:  2006-10-24     Completed Date:  2006-11-14     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  8112566     Medline TA:  J Cereb Blood Flow Metab     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1389-406     Citation Subset:  IM    
Affiliation:
Department of Neurosciences, School of Medicine, University of New Mexico, Albuquerque, New Mexico 87131, USA.
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MeSH Terms
Descriptor/Qualifier:
2-Amino-5-phosphonovalerate / pharmacology
6-Cyano-7-nitroquinoxaline-2,3-dione / pharmacology
Animals
Bicuculline / pharmacology
Brain / physiology*
Electric Stimulation
Excitatory Amino Acid Antagonists / pharmacology
Excitatory Postsynaptic Potentials / physiology
Flavoproteins / metabolism
Fluorescence
GABA Antagonists / pharmacology
Glutamic Acid / metabolism
Glycolysis / drug effects,  physiology
Long-Term Potentiation / drug effects
Male
Mice
Mice, Inbred C57BL
Mitochondria / physiology*
NADP / metabolism*
Neuroglia / physiology
Neurons / physiology
Pyruvates / metabolism
Synapses / physiology*
Grant Support
ID/Acronym/Agency:
NS43458/NS/NINDS NIH HHS; RR15636/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Excitatory Amino Acid Antagonists; 0/Flavoproteins; 0/GABA Antagonists; 0/Pyruvates; 115066-14-3/6-Cyano-7-nitroquinoxaline-2,3-dione; 485-49-4/Bicuculline; 53-59-8/NADP; 56-86-0/Glutamic Acid; 76726-92-6/2-Amino-5-phosphonovalerate

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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