Document Detail


N-linked glycosylation determines cell surface expression of two-pore-domain K+ channel TRESK.
MedLine Citation:
PMID:  20006580     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Within the first external loop of mouse and human TRESK subunits one or two N-glycosylation consensus sites were identified, respectively. Using site directed mutagenesis and Western immunoblotting a single residue of both orthologues was found to be glycosylated upon heterologous expression. Two-electrode voltage-clamp recordings from Xenopus oocytes revealed that current amplitudes of N-glycosylation mutants were reduced by 80% as compared to wildtype TRESK. To investigate membrane targeting, GFP-tagged TRESK subunits were expressed in Xenopus oocytes and fluorescence intensity at the cell surface was measured by confocal microscopy. Signals of the N-glycosylation mutants were reduced by >50%, indicating that their lower current amplitudes substantially result from inadequate surface expression of the channel.
Authors:
Brigitte Egenberger; Georg Polleichtner; Erhard Wischmeyer; Frank D?ring
Publication Detail:
Type:  Journal Article     Date:  2009-12-16
Journal Detail:
Title:  Biochemical and biophysical research communications     Volume:  391     ISSN:  1090-2104     ISO Abbreviation:  Biochem. Biophys. Res. Commun.     Publication Date:  2010 Jan 
Date Detail:
Created Date:  2010-01-27     Completed Date:  2010-03-08     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0372516     Medline TA:  Biochem Biophys Res Commun     Country:  United States    
Other Details:
Languages:  eng     Pagination:  1262-7     Citation Subset:  IM    
Copyright Information:
Copyright 2009 Elsevier Inc. All rights reserved.
Affiliation:
Institute of Physiology, Department of Neurophysiology, University of W?rzburg, 97070 W?rzburg, Germany.
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MeSH Terms
Descriptor/Qualifier:
Amino Acid Sequence
Animals
Cell Membrane / metabolism*
Glycosylation
Humans
Mice
Molecular Sequence Data
Oocytes
Patch-Clamp Techniques
Potassium Channels / genetics,  metabolism*
Xenopus
Chemical
Reg. No./Substance:
0/KCNK18 protein, human; 0/Potassium Channels; 0/Trik protein, mouse

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