Document Detail


Myocardial reperfusion injury in neuronal nitric oxide synthase deficient mice.
MedLine Citation:
PMID:  11107506     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: A substantial amount of data suggesting that endothelial cell nitric oxide synthase (eNOS) plays a cardioprotective role in animal models of ischemia-reperfusion injury has amassed. We have previously demonstrated that eNOS-deficient (-/-) mice exhibit significantly larger myocardial infarcts than do wild-type mice. Few investigations have examined the neuronal form of nitric oxide synthase in the heart. The two constitutive isoforms have been demonstrated to play differing roles in studies of cerebral ischemia-reperfusion. OBJECTIVE: To characterize the role of neuronal nitric oxide synthase (nNOS) in myocardial ischemia-reperfusion injury. METHODS: Wild-type and nNOS -/- mice were subjected to 20 min of coronary artery occlusion and 120 min of reflow. RESULTS: We found no significant difference between the two groups in terms of infarct size. Microscopic cross-sections from both groups were examined for infiltration of polymorphonuclear leukocyte. Hearts of nNOS -/- mice exhibited significantly (P < 0.05) more polymorphonuclear leukocytes than did hearts of wild-type mice. CONCLUSION: Despite the fact that eNOS plays a cardioprotective role in the ischemic-reperfused myocardium, we observed no change in size of myocardial infarcts when nNOS was genetically disrupted.
Authors:
S P Jones; W G Girod; P L Huang; D J Lefer
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Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Coronary artery disease     Volume:  11     ISSN:  0954-6928     ISO Abbreviation:  Coron. Artery Dis.     Publication Date:  2000 Dec 
Date Detail:
Created Date:  2001-02-21     Completed Date:  2001-03-29     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  9011445     Medline TA:  Coron Artery Dis     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  593-7     Citation Subset:  IM    
Affiliation:
Department of Molecular and Cellular Physiology, Louisiana State University Health Sciences Center, Shreveport 71130, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Male
Mice
Mice, Inbred C57BL
Mice, Transgenic
Myocardial Reperfusion Injury / etiology*,  physiopathology
Myocardium / pathology
Nerve Tissue Proteins / deficiency*,  physiology
Neutrophils / physiology
Nitric Oxide Synthase / deficiency*,  physiology
Nitric Oxide Synthase Type I
Grant Support
ID/Acronym/Agency:
P01 DK 43785/DK/NIDDK NIH HHS; R01 HL 60849/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Nerve Tissue Proteins; EC 1.14.13.39/Nitric Oxide Synthase; EC 1.14.13.39/Nitric Oxide Synthase Type I; EC 1.14.13.39/Nos1 protein, mouse

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