Document Detail


Myocardial lesions in experimental acute heart failure.
MedLine Citation:
PMID:  6239100     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The present study concerns the morphological and biochemical lesional picture of the myocardium in cases of acute heart failure induced by various experimental models: ligature of the coronary artery, direct electric stimulation of the heart by catheterism, lethal hemorrhage, pneumothorax, beta-adrenergic shock. Worthy of note was the similitude of the lesional myocardium pictures characterized electron microscopically by a wide range of lesions, from reversible to focal cytolysis, and biochemically by decrease of mitochondrial enzymes, ATP, Mg2-1 X K+ and increase of Na+ X H2O, Ca2+. Problems linked to the pathogenesis, reversibility of the lesions and efficiency of certain therapeutical means are discussed.
Authors:
D Laky; S Constantinescu; G Filipescu; E Ratea; F Hălălău
Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Morphologie et embryologie     Volume:  30     ISSN:  0377-5038     ISO Abbreviation:  Morphol Embryol (Bucur)     Publication Date:    1984 Jul-Sep
Date Detail:
Created Date:  1985-01-23     Completed Date:  1985-01-23     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  7512183     Medline TA:  Morphol Embryol (Bucur)     Country:  ROMANIA    
Other Details:
Languages:  eng     Pagination:  223-7     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Acute Disease
Animals
Dogs
Electrocardiography
Female
Guinea Pigs
Heart Failure / enzymology,  pathology*,  physiopathology
Male
Myocardium / pathology*,  ultrastructure
Rats

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Morphological aspects of the rat kidney preserved by cold storage. IV. Histoenzymological changes. V...
Next Document:  Recurrent venous thromboembolism in patients with a partial deficiency of protein S.