Document Detail


Myocardial contractile performance during acute coronary occlusion with special emphasis on nonoccluded regions.
MedLine Citation:
PMID:  1867109     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Ischaemia of the left ventricle caused by coronary artery occlusion generally leads to less changes in overall ventricular performance than would be expected from the amount of ischaemic, noncontracting tissue. Ischaemic myocardium shows elongation of segment lengths and paradoxial movement during contraction of the left ventricle. Compensation for the loss of contractile tissue is most likely related to increased performance of the nonischaemic myocardium and the most likely mechanisms involved are discussed. The effect of increased preload, brought about by increased left ventricular end-diastolic pressure following coronary occlusion, on nonischaemic parts appear to be the most important mechanism of compensation.
Authors:
E Hexeberg; S Birkeland; K Matre; J Lekven
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Acta physiologica Scandinavica. Supplementum     Volume:  599     ISSN:  0302-2994     ISO Abbreviation:  Acta Physiol Scand Suppl     Publication Date:  1991  
Date Detail:
Created Date:  1991-09-12     Completed Date:  1991-09-12     Revised Date:  2008-02-20    
Medline Journal Info:
Nlm Unique ID:  0376307     Medline TA:  Acta Physiol Scand Suppl     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  99-107     Citation Subset:  IM; S    
Affiliation:
Surgical Research Laboratory, Department of Surgery, University of Bergen.
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MeSH Terms
Descriptor/Qualifier:
Adaptation, Physiological
Animals
Blood Pressure
Coronary Disease / physiopathology*
Heart Rate
Myocardial Contraction*
Myocardium / pathology*
Regional Blood Flow
Ventricular Function, Left*

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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