Document Detail


Morphological changes and morphological-functional correlations in acute experimental ischemia/reperfusion pancreatitis in rats.
MedLine Citation:
PMID:  11247388     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The etiology of acute pancreatitis, apart from alcohol abuse and cholelithiasis may also include a vascular component responsible for pancreatic ischemia. It is now acknowledged that chronic pancreatitis may be a consequence of the acute variant, but it remains unclear what factors influence this sequence of morphological changes. In order to clarify this issue we proposed a model of experimental acute pancreatitis in rats induced by a 30 min reduction of blood flow in inferior splenic artery followed by reperfusion. Rats were sacrificed at 1 h, 12 h, 24 h, and 2, 3, 5, 7, 14, 21, 28 days after cessation of ischemia. We performed histopathological examination of pancreatic tissue and measured pancreatic blood flow, plasma amylase activity and interleukin-1 beta concentration. The present findings indicate that transient pancreatic ischemia leads to the development of acute necro-haemorrhagic pancreatitis. The morphological features of acute inflammation are correlated positively with functional disorders. In some cases the features of chronic pancreatitis may appear transiently after the acute phase, whereas the repair of postinflammatory injury involves the regeneration of acinar cells.
Authors:
R Tomaszewska; A Dembiński; Z Warzecha; P Ceranowicz; J Stachura
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Polish journal of pathology : official journal of the Polish Society of Pathologists     Volume:  51     ISSN:  1233-9687     ISO Abbreviation:  Pol J Pathol     Publication Date:  2000  
Date Detail:
Created Date:  2001-03-14     Completed Date:  2001-04-12     Revised Date:  2007-08-01    
Medline Journal Info:
Nlm Unique ID:  9437432     Medline TA:  Pol J Pathol     Country:  Poland    
Other Details:
Languages:  eng     Pagination:  179-84     Citation Subset:  IM    
Affiliation:
Department of Clinical and Experimental Pathomorphology, Jagiellonian University Medical College, Kraków.
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MeSH Terms
Descriptor/Qualifier:
Acute Disease
Amylases / blood
Animals
Interleukin-1 / blood
Ischemia / blood,  pathology
Male
Pancreas / blood supply,  enzymology
Pancreatitis / blood,  pathology*
Rats
Rats, Wistar
Regional Blood Flow
Reperfusion Injury / blood,  pathology*
Chemical
Reg. No./Substance:
0/Interleukin-1; EC 3.2.1.-/Amylases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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