Document Detail


Molecular mechanisms of pituitary organogenesis: In search of novel regulatory genes.
MedLine Citation:
PMID:  20025935     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Defects in pituitary gland organogenesis are sometimes associated with congenital anomalies that affect head development. Lesions in transcription factors and signaling pathways explain some of these developmental syndromes. Basic research studies, including the characterization of genetically engineered mice, provide a mechanistic framework for understanding how mutations create the clinical characteristics observed in patients. Defects in BMP, WNT, Notch, and FGF signaling pathways affect induction and growth of the pituitary primordium and other organ systems partly by altering the balance between signaling pathways. The PITX and LHX transcription factor families influence pituitary and head development and are clinically relevant. A few later-acting transcription factors have pituitary-specific effects, including PROP1, POU1F1 (PIT1), and TPIT (TBX19), while others, such as NeuroD1 and NR5A1 (SF1), are syndromic, influencing development of other endocrine organs. We conducted a survey of genes transcribed in developing mouse pituitary to find candidates for cases of pituitary hormone deficiency of unknown etiology. We identified numerous transcription factors that are members of gene families with roles in syndromic or non-syndromic pituitary hormone deficiency. This collection is a rich source for future basic and clinical studies.
Authors:
S W Davis; F Castinetti; L R Carvalho; B S Ellsworth; M A Potok; R H Lyons; M L Brinkmeier; L T Raetzman; P Carninci; A H Mortensen; Y Hayashizaki; I J P Arnhold; B B Mendonça; T Brue; S A Camper
Related Documents :
19281745 - Atypical alport syndrome associated with a novel col4a5 mutation.
8593545 - A tale of four syndromes: familial adenomatous polyposis, gardner syndrome, attenuated ...
11730045 - Waardenburg syndrome.
19376485 - Mutational screening of basp1 and transcribed processed pseudogene tppsig-basp1 in pati...
2144945 - Atrioventricular canal in down syndrome. prevalence of associated cardiac malformations...
22140075 - Myelodysplastic syndrome in a child with 15q24 deletion syndrome.
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2009-12-16
Journal Detail:
Title:  Molecular and cellular endocrinology     Volume:  323     ISSN:  1872-8057     ISO Abbreviation:  Mol. Cell. Endocrinol.     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-05-20     Completed Date:  2010-08-20     Revised Date:  2011-08-01    
Medline Journal Info:
Nlm Unique ID:  7500844     Medline TA:  Mol Cell Endocrinol     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  4-19     Citation Subset:  IM    
Affiliation:
University of Michigan Medical School, Ann Arbor, MI 41809-5618, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Cell Communication / genetics,  physiology
Female
Gene Expression Regulation, Developmental*
Genes, Developmental*
Homeodomain Proteins / genetics,  physiology
Human Growth Hormone / deficiency,  physiology
Humans
Male
Mice
Organogenesis / genetics*
Pituitary Gland / growth & development*
Transcription Factors / genetics,  physiology
Grant Support
ID/Acronym/Agency:
NRSA F32-HD046300/HD/NICHD NIH HHS; R01 HD034283-14/HD/NICHD NIH HHS; R01 HD034283-15/HD/NICHD NIH HHS; R01HD034283/HD/NICHD NIH HHS; R37 HD030428-17/HD/NICHD NIH HHS; R37 HD030428-19/HD/NICHD NIH HHS; R37HD030428/HD/NICHD NIH HHS; T32 HD07048/HD/NICHD NIH HHS
Chemical
Reg. No./Substance:
0/Homeodomain Proteins; 0/Transcription Factors; 12629-01-5/Human Growth Hormone
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  NELF is a nuclear protein involved in hypothalamic GnRH neuronal migration.
Next Document:  The development and endocrine functions of adipose tissue.