Document Detail


Modulation of blood pressure by substance P: opposite effects of N- and C- terminal fragments on anesthetized rats.
MedLine Citation:
PMID:  2437424     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Considerable evidence suggests that substance P (SP) may be a transmitter mediating the depressor effects of baroreceptor reflex activation within the brainstem, yet intracerebroventricular administration of SP is reported to result in a pronounced pressor effect. In this study, SP injected into the 4th cerebral ventricle produced a biphasic effect; a brief decrease in blood pressure followed by a lengthy increase. Similar injections of a carboxy-terminal fragment of SP produced only a pressor effect of long duration. Injection of an amino-terminal SP fragment produced only a brief, rapid depressor effect. These results suggest that the amino-terminal sequence of SP may be involved in mediating the depressor effects of baroreceptor activation.
Authors:
M E Hall; F B Miley; J M Stewart
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Life sciences     Volume:  40     ISSN:  0024-3205     ISO Abbreviation:  Life Sci.     Publication Date:  1987 May 
Date Detail:
Created Date:  1987-06-18     Completed Date:  1987-06-18     Revised Date:  2007-11-14    
Medline Journal Info:
Nlm Unique ID:  0375521     Medline TA:  Life Sci     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  1909-14     Citation Subset:  IM    
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MeSH Terms
Descriptor/Qualifier:
Anesthesia
Animals
Blood Pressure / drug effects*
Male
Peptide Fragments / pharmacology*
Pressoreceptors / drug effects,  physiology
Rats
Rats, Inbred Strains
Substance P / pharmacology*
Time Factors
Grant Support
ID/Acronym/Agency:
HL 33534/HL/NHLBI NIH HHS
Chemical
Reg. No./Substance:
0/Peptide Fragments; 33507-63-0/Substance P; 72226-88-1/substance P (1-7)

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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