Document Detail


Modulating heme redox potential through protein-induced porphyrin distortion.
MedLine Citation:
PMID:  20735135     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hemoproteins are ubiquitous in biology and are commonly involved in critical processes such as electron transfer, oxidative phosphorylation, and signal transduction. Both the protein environment and the heme cofactor contribute to generate the range of chemical properties needed for these diverse functions. Using the heme nitric oxide/oxygen binding (H-NOX) protein from the thermophilic bacterium Thermoanaerobacter tengcongensis, we have shown that heme electronic properties can be modulated by porphyrin distortion within the same protein scaffold without changing the heme ligation state or heme environment. The degree of heme distortion was found to be directly correlated to the electron density at the heme iron, as evidenced by dramatic changes in the heme redox potential and pK(a) of the distal ligand ((-)OH vs H(2)O). Protein-induced porphyrin distortion represents a new strategy to rationally tune the electronic properties of protein-bound porphyrins and could be used to engineer proteins with desired reactivity or functionality.
Authors:
Charles Olea; John Kuriyan; Michael A Marletta
Related Documents :
16771505 - Redox-coupled dynamics and folding in cytochrome c.
173545 - Nitrogenase from azotobacter chroococcum. purification and properties of the component ...
15651045 - A new type of metalloprotein: the mo storage protein from azotobacter vinelandii contai...
10713545 - Cytochrome p450 1a2: oligomers in proteoliposomes.
19167355 - Immobilized metal affinity chromatography in the presence of arginine.
8792085 - Vav: captain hook for signal transduction?
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  Journal of the American Chemical Society     Volume:  132     ISSN:  1520-5126     ISO Abbreviation:  J. Am. Chem. Soc.     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-09-15     Completed Date:  2011-01-04     Revised Date:  2012-04-26    
Medline Journal Info:
Nlm Unique ID:  7503056     Medline TA:  J Am Chem Soc     Country:  United States    
Other Details:
Languages:  eng     Pagination:  12794-5     Citation Subset:  IM    
Affiliation:
Department of Molecular and Cell Biology, California Institute for Quantitative Biosciences, and Division of Physical Biosciences, University of California, Berkeley, Berkeley, California 94720-3220, USA.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Bacterial Proteins / chemistry*,  genetics,  metabolism*
Heme / chemistry*,  metabolism*
Hydrophobic and Hydrophilic Interactions
Ligands
Models, Molecular
Molecular Conformation*
Mutagenesis, Site-Directed
Oxidation-Reduction
Thermoanaerobacter
Grant Support
ID/Acronym/Agency:
GM070671/GM/NIGMS NIH HHS; R01 GM070671-04/GM/NIGMS NIH HHS
Chemical
Reg. No./Substance:
0/Bacterial Proteins; 0/Ligands; 14875-96-8/Heme
Comments/Corrections

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Tricyclic host for linear anions.
Next Document:  4D Lorentz electron microscopy imaging: magnetic domain wall nucleation, reversal, and wave velocity...