Document Detail


Modeling mechanisms of cell secretion.
MedLine Citation:
PMID:  20661627     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Secretion is a fundamental cellular process involving the regulated release of intracellular products from cells. Physiological functions such as neurotransmission, or the release of hormones and digestive enzymes, are all governed by cell secretion. Anomalies in the processes involved in secretion contribute to the development and progression of diseases such as diabetes and other hormonal disorders. To unravel the mechanisms that govern such diseases, it is essential to understand how hormones, growth factors and neurotransmitters are synthesized and processed, and how their signals are recognized, amplified and transmitted by intracellular signaling pathways in the target cells. Here, we discuss diverse aspects of the detailed mechanisms involved in secretion based on mathematical models. The models range from stochastic ones describing the trafficking of secretory vesicles to deterministic ones investigating the regulation of cellular processes that underlie hormonal secretion. In all cases, the models are closely related to experimental results and suggest theoretical predictions for the secretion mechanisms.
Authors:
Krasimira Tsaneva-Atanasova; Hinke M Osinga; Joël Tabak; Morten Gram Pedersen
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2010-07-27
Journal Detail:
Title:  Acta biotheoretica     Volume:  58     ISSN:  1572-8358     ISO Abbreviation:  Acta Biotheor.     Publication Date:  2010 Dec 
Date Detail:
Created Date:  2010-11-12     Completed Date:  2011-01-28     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0421520     Medline TA:  Acta Biotheor     Country:  Netherlands    
Other Details:
Languages:  eng     Pagination:  315-27     Citation Subset:  IM    
Affiliation:
Bristol Centre for Applied Nonlinear Mathematics, Department of Engineering Mathematics, University of Bristol, Queen's Building, University Walk, Bristol BS8 1TR, UK. K.Tsaneva-Atanasova@bristol.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Animals
Biological Transport*
Calcium / metabolism
Cell Physiological Phenomena*
Exocytosis*
Hormones / secretion*
Humans
Models, Biological
Secretory Vesicles / metabolism
Signal Transduction
Grant Support
ID/Acronym/Agency:
DA-19356/DA/NIDA NIH HHS
Chemical
Reg. No./Substance:
0/Hormones; 7440-70-2/Calcium

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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