Document Detail


Mitochondrial structure and function in acute myocardial ischemic injury.
MedLine Citation:
PMID:  1269093     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Changes in both the structure and function of mitochondria occur in the dog heart as a consequence of severe ischemia produced by acute coronary occlusion. Brief periods of severe ischemia (reversible injury) produced no significant change in mitochondrial ultrastructure and no defects in pyruvate or succinate metabolism. However, periods of ischemia of 40-60 minutes' duration (irreversible injury) produced striking structural changes including swelling, an increase in matrix space, disorganization of cristae, and the appearance of amorphous matrix densities. After 60 minutes of severe ischemia, one or more amorphous densities were present in each mitochondrial profile. These osmiophilic structures contained lipid but have not been characterized further. Their presence was typical of the irreversible state. Mitochondria of irreversibly injured cells were fragile, and consequently were more difficult to isolate than mitochondria of control tissue. Furthermore, after isolation from tissue injured by 60 minutes of ischemia, they showed markedly defective function.
Authors:
R B Jennings; C E Ganote
Related Documents :
9274853 - Prevention of cerebral edema and infarct in cerebral reperfusion injury by an antibody ...
2560953 - Prevention of myocardial reperfusion injury with free radical scavengers. an experiment...
9926273 - Gallopamil activity on asynergic viable myocardium in acute myocardial infarction: insi...
Publication Detail:
Type:  In Vitro; Journal Article; Research Support, U.S. Gov't, P.H.S.    
Journal Detail:
Title:  Circulation research     Volume:  38     ISSN:  0009-7330     ISO Abbreviation:  Circ. Res.     Publication Date:  1976 May 
Date Detail:
Created Date:  1976-08-02     Completed Date:  1976-08-02     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0047103     Medline TA:  Circ Res     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  I80-91     Citation Subset:  IM    
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Animals
Coronary Disease / pathology*,  physiopathology
Disease Models, Animal
Dogs
Mitochondria, Muscle / metabolism,  ultrastructure*
Mitochondrial Swelling
Myocardium / metabolism,  ultrastructure*
Oxygen Consumption
Pyruvates / metabolism
Succinate Dehydrogenase / metabolism
Succinates / metabolism
Chemical
Reg. No./Substance:
0/Pyruvates; 0/Succinates; EC 1.3.99.1/Succinate Dehydrogenase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Control of energy production in myocardial ischemia.
Next Document:  Considerations in the use of biochemical markers of ischemic injury.