Document Detail


Mitochondrial dysfunction: impact on exercise performance and cellular aging.
MedLine Citation:
PMID:  17417049     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Innovative noninvasive methods open a new window on the cell in vivo. This window reveals that the tempo of mitochondrial dysfunction with age varies among muscles and in proportion to Type II muscle fiber content. Exercise training can reverse age-related dysfunction, thereby providing an intervention to slow the pace of aging and disability in the elderly.
Authors:
Kevin E Conley; Sharon A Jubrias; Catherine E Amara; David J Marcinek
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Review    
Journal Detail:
Title:  Exercise and sport sciences reviews     Volume:  35     ISSN:  0091-6331     ISO Abbreviation:  Exerc Sport Sci Rev     Publication Date:  2007 Apr 
Date Detail:
Created Date:  2007-04-09     Completed Date:  2007-08-27     Revised Date:  2007-12-03    
Medline Journal Info:
Nlm Unique ID:  0375434     Medline TA:  Exerc Sport Sci Rev     Country:  United States    
Other Details:
Languages:  eng     Pagination:  43-9     Citation Subset:  IM    
Affiliation:
Department of Radiology, University of Washington Medical Center, Seattle, WA 98195-7115, USA. kconley@u.washington.edu
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MeSH Terms
Descriptor/Qualifier:
Adult
Cell Aging / physiology*
DNA, Mitochondrial
Exercise / physiology*
Humans
Mitochondria, Muscle / pathology,  physiology*
Muscle, Skeletal / pathology,  physiology*
Sports Medicine
Grant Support
ID/Acronym/Agency:
R01 AG 00057/AG/NIA NIH HHS; R01 AG 022385/AG/NIA NIH HHS; R01 AR 36281/AR/NIAMS NIH HHS; R01 AR 41928/AR/NIAMS NIH HHS; R01 AR 45184/AR/NIAMS NIH HHS
Chemical
Reg. No./Substance:
0/DNA, Mitochondrial

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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