Document Detail


Mineralocorticoid hypertension.
MedLine Citation:
PMID:  10218547     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Hypertension with hypokalaemia and suppression of plasma renin activity is known as mineralocorticoid hypertension. Although mineralocorticoid hypertension accounts for a small number of patients labelled as having "essential" hypertension, it is a potentially reversible cause of high blood pressure. The most common cause of mineralocorticoid hypertension is probably primary aldosteronism; controlled posture studies to measure plasma renin activity and aldosterone concentrations, followed by adrenal imaging, will ensure the differential diagnosis between an aldosterone-producing adenoma and idiopathic adrenal hyperplasia in most cases. Three monogenic forms of mineralocorticoid hypertension have been described: glucocorticoid-suppressible hyperaldosteronism, Liddle's syndrome, and apparent mineralocorticoid excess, which have provided new insights into mineralocorticoid hormone action. Many patients with mineralocorticoid-based hypertension are now known to have normal serum potassium concentrations. Until the true prevalence of primary aldosteronism and monogenic forms of mineralocorticoid hypertension are defined, a high index of suspicion is needed in every hypertensive patient. Hypertensive patients with hypokalaemia, together with those with severe hypertension or a family history of hypertension or stroke, should be screened for mineralocorticoid excess.
Authors:
P M Stewart
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Lancet     Volume:  353     ISSN:  0140-6736     ISO Abbreviation:  Lancet     Publication Date:  1999 Apr 
Date Detail:
Created Date:  1999-05-20     Completed Date:  1999-05-20     Revised Date:  2005-11-16    
Medline Journal Info:
Nlm Unique ID:  2985213R     Medline TA:  Lancet     Country:  ENGLAND    
Other Details:
Languages:  eng     Pagination:  1341-7     Citation Subset:  AIM; IM    
Affiliation:
Department of Medicine, Queen Elizabeth Hospital, Edgbaston, Birmingham, UK. p.m.stewart@bham.ac.uk
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MeSH Terms
Descriptor/Qualifier:
Algorithms
Humans
Hyperaldosteronism / epidemiology,  genetics*
Hypertension / epidemiology,  etiology*,  genetics
Hypokalemia / genetics
Mineralocorticoids* / secretion
Prevalence
Chemical
Reg. No./Substance:
0/Mineralocorticoids

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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