Document Detail


Mild stress facilitates learning and exercise improves retention in aged mice.
MedLine Citation:
PMID:  20951791     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
While chronic or severe stress generally has negative consequences in the brain, moderate stress has been shown to facilitate learning in young animals. It is unknown if stress facilitates or impairs learning in an aged animal. In this study, the effect of mild stress on learning and task retention (1 week later) was assessed in young and aged mice. In addition, because exercise can counteract negative effects of stress and facilitate learning, the effect of 1 week of voluntary exercise on task retention was assessed. Regulation of the learning/memory related genes BDNF exon VI, synapsin, MAPK, and CRFR1 were also examined in the hippocampus using real-time PCR. Results of this study demonstrate that mild stress 24h prior to the learning test improved performance in aged, but not young animals. However, exercise for 1 week between tests improved the performance of both groups of animals in the retention task. In aged, but not young animals, all four genes increased significantly after exercise. Thus, in aged animals mild stress facilitates learning and the retention of this task is improved by voluntary exercise.
Authors:
Paul A Adlard; Christie Engesser-Cesar; Carl W Cotman
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-10-15
Journal Detail:
Title:  Experimental gerontology     Volume:  46     ISSN:  1873-6815     ISO Abbreviation:  Exp. Gerontol.     Publication Date:  2011 Jan 
Date Detail:
Created Date:  2010-12-07     Completed Date:  2011-03-25     Revised Date:  2011-10-13    
Medline Journal Info:
Nlm Unique ID:  0047061     Medline TA:  Exp Gerontol     Country:  England    
Other Details:
Languages:  eng     Pagination:  53-9     Citation Subset:  IM    
Copyright Information:
Copyright © 2010 Elsevier Inc. All rights reserved.
Affiliation:
Institute for Brain Aging and Dementia, University of California Irvine, 1226 Gillespie NRF, Irvine, CA 92697-4540, United States. p.adlard@mhri.edu.au
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Age Factors
Aging / physiology*
Animals
Avoidance Learning / physiology*
Brain-Derived Neurotrophic Factor / genetics
Conditioning (Psychology) / physiology
Electroshock
Gene Expression / physiology
Male
Memory / physiology*
Mice
Mice, Inbred C57BL
Mitogen-Activated Protein Kinases / genetics
Physical Conditioning, Animal / physiology*
Receptors, Corticotropin-Releasing Hormone / genetics
Stress, Psychological / physiopathology*
Synapsins / genetics
Chemical
Reg. No./Substance:
0/Brain-Derived Neurotrophic Factor; 0/CRF receptor type 1; 0/Receptors, Corticotropin-Releasing Hormone; 0/Synapsins; EC 2.7.11.24/Mitogen-Activated Protein Kinases
Comments/Corrections
Erratum In:
Exp Gerontol. 2011 Oct;46(10):841

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Effects of chronic fentanyl administration on physical performance of aged rats.
Next Document:  IL-23 modulated myelin-specific T cells induce EAE via an IFN? driven, IL-17 independent pathway.