Document Detail


Microphthalmia-associated transcription factor controls the DNA damage response and a lineage-specific senescence program in melanomas.
MedLine Citation:
PMID:  20388797     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Apoptosis and senescence are cellular failsafe programs that counteract excessive mitogenic signaling observed in cancer cells. Melanoma is known for its notorious resistance to apoptotic processes; therefore, senescence, which remains poorly understood in melanomas, can be viewed as a therapeutic alternative. Microphthalmia-associated transcription factor (MITF), in which its M transcript is specifically expressed in melanocyte cells, plays a critical role in melanoma proliferation, and its specific inhibition is associated with G(0)-G(1) growth arrest. Interestingly, decreased MITF expression has been described in senescent melanocytes, and we have observed an inhibition of MITF expression in melanoma cells exposed to chemotherapeutic drugs that induce their senescence. All these observations thereby question the role of MITF in controlling senescence in melanoma cells. Here, we report that long-term depletion of MITF in melanoma cells triggers a senescence program characterized by typical morphologic and biochemical changes associated with a sustained growth arrest. Further, we show that MITF-silenced cells engage a DNA damage response (DDR) signaling pathway, leading to p53 upregulation, which is critically required for senescence entry. This study uncovers the existence of a lineage-restricted DDR/p53 signaling pathway that is inhibited by MITF to prevent senescence and favor melanoma cell proliferation.
Authors:
Sandy Giuliano; Yann Cheli; Micka?l Ohanna; Caroline Bonet; Laurent Beuret; Karine Bille; Agn?s Loubat; V?ronique Hofman; Paul Hofman; Gilles Ponzio; Philippe Bahadoran; Robert Ballotti; Corine Bertolotto
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-04-13
Journal Detail:
Title:  Cancer research     Volume:  70     ISSN:  1538-7445     ISO Abbreviation:  Cancer Res.     Publication Date:  2010 May 
Date Detail:
Created Date:  2010-04-30     Completed Date:  2010-05-27     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  2984705R     Medline TA:  Cancer Res     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3813-22     Citation Subset:  IM    
Copyright Information:
(c)2010 AACR.
Affiliation:
Institut National de la Sante et de la Recherche Medicale U895, University of Nice Sophia-Antipolis, Nice, France.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cell Aging / physiology
Cell Line, Tumor
Cell Lineage / physiology
DNA Damage*
Humans
Melanoma / genetics*,  metabolism,  pathology
Melanoma, Experimental / genetics,  metabolism,  pathology
Mice
Microphthalmia-Associated Transcription Factor / deficiency*,  genetics,  metabolism
Mitosis / genetics
Signal Transduction
Tumor Suppressor Protein p53 / genetics,  metabolism
Chemical
Reg. No./Substance:
0/Microphthalmia-Associated Transcription Factor; 0/Tumor Suppressor Protein p53

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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