Document Detail


Methotrexate-induced senescence in human adenocarcinoma cells is accompanied by induction of p21(waf1/cip1) expression and lack of polyploidy.
MedLine Citation:
PMID:  19467772     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Human colorectal adenocarcinoma C85 cells, treated with high dose methotrexate (1 microM; IC(50)=51 nM), undergo accelerated senescence, as the cells (i) are growth arrested at the G(1) and S phases of the cell cycle, (ii) are SA-beta-galactosidase-positive, (iii) show induced expression of p21(waf1/cip1) and decreased expression of p16(INK4a), and (iv) show DNA synthesis continued at the reduced level. The fraction of C85 cells with DNA content higher than 4N is maintained at the same level (14%) in cells untreated, as well as regrown after the treatment. Multinucleation is found as the main karyotypic abnormality.
Authors:
Magdalena Dabrowska; Grazyna Mosieniak; Janusz Skierski; Ewa Sikora; Wojciech Rode
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2009-05-20
Journal Detail:
Title:  Cancer letters     Volume:  284     ISSN:  1872-7980     ISO Abbreviation:  Cancer Lett.     Publication Date:  2009 Oct 
Date Detail:
Created Date:  2009-08-31     Completed Date:  2009-09-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  7600053     Medline TA:  Cancer Lett     Country:  Ireland    
Other Details:
Languages:  eng     Pagination:  95-101     Citation Subset:  IM    
Affiliation:
Nencki Institute of Experimental Biology, Polish Academy of Sciences, 3 Pasteur St., Warsaw 02-093, Poland. m.dabrowska@nencki.gov.pl
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MeSH Terms
Descriptor/Qualifier:
Antimetabolites, Antineoplastic / pharmacology*
Cell Aging
Cell Cycle / drug effects,  physiology*
Cell Line, Tumor
Cell Survival / drug effects,  physiology
Cyclin-Dependent Kinase Inhibitor p16 / metabolism
Cyclin-Dependent Kinase Inhibitor p21 / metabolism*
Humans
Methotrexate / pharmacology*
Polyploidy*
Chemical
Reg. No./Substance:
0/Antimetabolites, Antineoplastic; 0/Cyclin-Dependent Kinase Inhibitor p16; 0/Cyclin-Dependent Kinase Inhibitor p21; 59-05-2/Methotrexate

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