Document Detail


Metabolic effects of infection and postnatal steroids.
MedLine Citation:
PMID:  12168243     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Optimal development of the newborn depends on rapid accretion of substrate in the neonatal period, particularly in the premature infant. Steroids and infection not only induce catabolism, but associated endogenous responses reprioritize crucial substrate to restore homeostasis. The result is a protein/energy deficit and concomitant delay in growth and development. Innovative feeding strategies and novel therapies are needed to reduce the impact of catabolism in this population.
Authors:
Sarah Jane Schwarzenberg; Andras Kovacs
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Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Clinics in perinatology     Volume:  29     ISSN:  0095-5108     ISO Abbreviation:  Clin Perinatol     Publication Date:  2002 Jun 
Date Detail:
Created Date:  2002-08-09     Completed Date:  2003-01-23     Revised Date:  2008-11-21    
Medline Journal Info:
Nlm Unique ID:  7501306     Medline TA:  Clin Perinatol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  295-312     Citation Subset:  IM    
Affiliation:
Department of Pediatrics, University of Minnesota, MMC 185, Minneapolis, MN 55455, USA. schwa005@tc.umn.edu
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MeSH Terms
Descriptor/Qualifier:
Child Development / drug effects
Energy Metabolism* / drug effects
Glucocorticoids / adverse effects,  therapeutic use*
Humans
Infant Nutritional Physiological Phenomena*
Infant, Newborn
Infant, Premature, Diseases / drug therapy
Infection / metabolism*
Lipid Metabolism*
Micronutrients / metabolism
Nutritional Requirements
Proteins / metabolism*
Sepsis / metabolism
Chemical
Reg. No./Substance:
0/Glucocorticoids; 0/Micronutrients; 0/Proteins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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