| Metabolic dysfunction in the pathogenesis of pulmonary hypertension. | |
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MedLine Citation:
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PMID: 20889122 Owner: NLM Status: In-Process |
Abstract/OtherAbstract:
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Pulmonary artery hypertension is characterized by proliferation in the resistance vessels. A recent study in Science Translational Medicine (Sutendra et al., 2010) found that increased fatty acid oxidation and a shift in the glycolysis/glucose oxidation ratio may be central to the pathogenesis of this process, suggesting that these abnormalities comprise novel treatment targets. |
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Authors:
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Lewis J Rubin |
Publication Detail:
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Type: Comment; Journal Article |
Journal Detail:
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Title: Cell metabolism Volume: 12 ISSN: 1932-7420 ISO Abbreviation: Cell Metab. Publication Date: 2010 Oct |
Date Detail:
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Created Date: 2010-10-04 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 101233170 Medline TA: Cell Metab Country: United States |
Other Details:
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Languages: eng Pagination: 313-4 Citation Subset: - |
Copyright Information:
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Copyright © 2010 Elsevier Inc. All rights reserved. |
Affiliation:
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University of California, San Diego, La Jolla, CA 92093, USA. ljrubin@ucsd.edu |
Export Citation:
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| MeSH Terms | |
Descriptor/Qualifier:
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| Comments/Corrections | |
Comment On:
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Sci Transl Med. 2010 Aug 11;2(44):44ra58
[PMID:
20702857
]
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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