Document Detail


Metabolic dysfunction associated with adiponectin deficiency enhances kainic acid-induced seizure severity.
MedLine Citation:
PMID:  21976521     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Metabolic syndrome has deleterious effects on the CNS, and recent evidence suggests that obesity rates are higher at presentation in children who develop epilepsy. Adiponectin is secreted by adipose tissue and acts in the brain and peripheral organs to regulate glucose and lipid metabolism. Adiponectin deficiency predisposes toward metabolic syndrome, characterized by obesity, insulin resistance, impaired glucose tolerance, hyperlipidemia, and cardiovascular morbidity. To investigate the relationship between metabolic syndrome and seizures, wild-type C57BL/6J and adiponectin knock-out mice were fed a high-fat diet, followed by treatment with low doses of kainic acid to induce seizures. Adiponectin deficiency in mice fed a high-fat diet resulted in greater fat accumulation, impaired glucose tolerance, hyperlipidemia, increased seizure severity, and increased hippocampal pathology. In contrast, there were no adverse effects of adiponectin deficiency on metabolic phenotype or seizure activity in mice fed a normal (low-fat) chow diet. These findings demonstrate that metabolic syndrome modulates the outcome of seizures and brain injury.
Authors:
Edward B Lee; Genevieve Warmann; Ravindra Dhir; Rexford S Ahima
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural    
Journal Detail:
Title:  The Journal of neuroscience : the official journal of the Society for Neuroscience     Volume:  31     ISSN:  1529-2401     ISO Abbreviation:  J. Neurosci.     Publication Date:  2011 Oct 
Date Detail:
Created Date:  2011-10-06     Completed Date:  2011-11-18     Revised Date:  2012-04-09    
Medline Journal Info:
Nlm Unique ID:  8102140     Medline TA:  J Neurosci     Country:  United States    
Other Details:
Languages:  eng     Pagination:  14361-6     Citation Subset:  IM    
Affiliation:
Department of Medicine, Division of Endocrinology, Diabetes and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania 19104, USA.
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MeSH Terms
Descriptor/Qualifier:
Adiponectin / deficiency*
Adipose Tissue / metabolism
Animals
Blood Glucose / metabolism
Hippocampus / metabolism,  pathology
Kainic Acid / toxicity*
Metabolic Syndrome X / metabolism*,  pathology
Mice
Mice, Inbred C57BL
Mice, Knockout
Seizures / chemically induced,  metabolism*,  pathology
Severity of Illness Index*
Grant Support
ID/Acronym/Agency:
K08 AG039510-01/AG/NIA NIH HHS; K08-AG039510/AG/NIA NIH HHS; P01 DK049210-14/DK/NIDDK NIH HHS; P01-DK-049210/DK/NIDDK NIH HHS; P30 DK019525-34/DK/NIDDK NIH HHS; P30-DK19525/DK/NIDDK NIH HHS; R01 DK062348-08/DK/NIDDK NIH HHS; R01-DK-062348/DK/NIDDK NIH HHS; T32 AG000255-10/AG/NIA NIH HHS; T32-AG00255/AG/NIA NIH HHS
Chemical
Reg. No./Substance:
0/Adiponectin; 0/Blood Glucose; 487-79-6/Kainic Acid

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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