Document Detail


Metabolic changes in a pyruvate kinase gene deletion mutant of Corynebacterium glutamicum ATCC 13032.
MedLine Citation:
PMID:  20144730     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
To investigate primary effects of a pyruvate kinase (PYK) defect on glucose metabolism in Corynebacterium glutamicum, a pyk-deleted mutant was derived from wild-type C. glutamicum ATCC13032 using the double-crossover chromosome replacement technique. The mutant was then evaluated under glutamic acid-producing conditions induced by biotin limitation. The mutant showed an increased specific rate of glucose consumption, decreased growth, higher glutamic acid production, and aspartic acid formation during the glutamic acid production phase. A significant increase in phosphoenolpyruvate (PEP) carboxylase activity and a significant decrease in PEP carboxykinase activity occurred in the mutant, which suggested an enhanced overall flux of the anaplerotic pathway from PEP to oxaloacetic acid in the mutant. The enhanced anaplerotic flux may explain both the increased rate of glucose consumption and the higher productivity of glutamic acid in the mutant. Since the pyk-complemented strain had similar metabolic profiles to the wild-type strain, the observed changes represented intrinsic effects of pyk deletion on the physiology of C. glutamicum.
Authors:
Kazunori Sawada; Susumu Zen-in; Masaru Wada; Atsushi Yokota
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-02-06
Journal Detail:
Title:  Metabolic engineering     Volume:  12     ISSN:  1096-7184     ISO Abbreviation:  Metab. Eng.     Publication Date:  2010 Jul 
Date Detail:
Created Date:  2010-08-04     Completed Date:  2010-11-23     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  9815657     Medline TA:  Metab Eng     Country:  Belgium    
Other Details:
Languages:  eng     Pagination:  401-7     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Laboratory of Microbial Physiology, Research Faculty of Agriculture, Hokkaido University, Kita-ku, Sapporo, Hokkaido 060-8589, Japan.
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MeSH Terms
Descriptor/Qualifier:
Aspartic Acid / biosynthesis
Biotin / metabolism
Corynebacterium glutamicum / enzymology*,  genetics*
Glucose / metabolism
Glutamic Acid / biosynthesis
Metabolic Networks and Pathways*
Oxaloacetate / analysis
Phosphoenolpyruvate Carboxykinase (ATP) / analysis
Phosphoenolpyruvate Carboxylase / analysis
Pyruvate Kinase / genetics*,  metabolism*
Sequence Deletion
Chemical
Reg. No./Substance:
0/Oxaloacetate; 50-99-7/Glucose; 56-84-8/Aspartic Acid; 56-86-0/Glutamic Acid; 58-85-5/Biotin; EC 2.7.1.40/Pyruvate Kinase; EC 4.1.1.31/Phosphoenolpyruvate Carboxylase; EC 4.1.1.49/Phosphoenolpyruvate Carboxykinase (ATP)

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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