| Metabolic acidosis and bone disease. | |
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MedLine Citation:
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PMID: 8202056 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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Bone is the largest repository base in the body. Prolonged acid retention requires continuous buffering of acid if the organism is to survive. This paper discusses the role of bone buffering under both normal and altered conditions of acid-base homeostasis. We place particular emphasis on the roles of PTH and vitamin D in this process. We also discuss bone abnormalities in renal tubular acidosis and chronic renal failure. |
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Authors:
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S Eiam-ong; N A Kurtzman |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Mineral and electrolyte metabolism Volume: 20 ISSN: 0378-0392 ISO Abbreviation: Miner Electrolyte Metab Publication Date: 1994 |
Date Detail:
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Created Date: 1994-07-07 Completed Date: 1994-07-07 Revised Date: 2005-11-16 |
Medline Journal Info:
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Nlm Unique ID: 7802196 Medline TA: Miner Electrolyte Metab Country: SWITZERLAND |
Other Details:
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Languages: eng Pagination: 72-80 Citation Subset: IM |
Affiliation:
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Department of Internal Medicine, Texas Tech University Health Sciences Center, Lubbock 79430. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Acidosis
/
complications,
metabolism* Acidosis, Renal Tubular / complications, metabolism Animals Bone Diseases, Metabolic / complications* Buffers Humans Kidney Failure, Chronic / complications Renal Osteodystrophy / complications, therapy |
| Chemical | |
Reg. No./Substance:
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0/Buffers |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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