Document Detail


Metabolic acidosis and bone disease.
MedLine Citation:
PMID:  8202056     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Bone is the largest repository base in the body. Prolonged acid retention requires continuous buffering of acid if the organism is to survive. This paper discusses the role of bone buffering under both normal and altered conditions of acid-base homeostasis. We place particular emphasis on the roles of PTH and vitamin D in this process. We also discuss bone abnormalities in renal tubular acidosis and chronic renal failure.
Authors:
S Eiam-ong; N A Kurtzman
Related Documents :
15577236 - Quantitative taste evaluation of total enteral nutrients.
18968846 - Determination of the total acidity in soft drinks using potentiometric sequential injec...
10795826 - Plastid genome phylogeny and a model of amino acid substitution for proteins encoded by...
18967966 - Titration of strong and weak acids by sequential injection analysis technique.
9168456 - Effect of docosahexaenoic acid on mouse mitochondrial membrane properties.
2822136 - The effect of ionophore a23187, verapamil, and dibutyryl cyclic amp on bile acid synthe...
Publication Detail:
Type:  Journal Article; Review    
Journal Detail:
Title:  Mineral and electrolyte metabolism     Volume:  20     ISSN:  0378-0392     ISO Abbreviation:  Miner Electrolyte Metab     Publication Date:  1994  
Date Detail:
Created Date:  1994-07-07     Completed Date:  1994-07-07     Revised Date:  2005-11-16    
Medline Journal Info:
Nlm Unique ID:  7802196     Medline TA:  Miner Electrolyte Metab     Country:  SWITZERLAND    
Other Details:
Languages:  eng     Pagination:  72-80     Citation Subset:  IM    
Affiliation:
Department of Internal Medicine, Texas Tech University Health Sciences Center, Lubbock 79430.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Acidosis / complications,  metabolism*
Acidosis, Renal Tubular / complications,  metabolism
Animals
Bone Diseases, Metabolic / complications*
Buffers
Humans
Kidney Failure, Chronic / complications
Renal Osteodystrophy / complications,  therapy
Chemical
Reg. No./Substance:
0/Buffers

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  The physicochemical structure of bone: cellular and noncellular elements.
Next Document:  The role of chronic anion gap and/or nonanion gap acidosis in the osteodystrophy of chronic renal fa...