Document Detail


Metabolic profiling of hearts exposed to sevoflurane and propofol reveals distinct regulation of fatty acid and glucose oxidation: CD36 and pyruvate dehydrogenase as key regulators in anesthetic-induced fuel shift.
MedLine Citation:
PMID:  20683255     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
BACKGROUND: Myocardial energy metabolism is a strong predictor of postoperative cardiac function. This study profiled the metabolites and metabolic changes in the myocardium exposed to sevoflurane, propofol, and Intralipid and investigated the underlying molecular mechanisms. METHODS: Sevoflurane (2 vol%) and propofol (10 and 100 microM) in the formulation of 1% Diprivan (AstraZeneca Inc., Mississauga, ON, Canada) were compared for their effects on oxidative energy metabolism and contractility in the isolated working rat heart model. Intralipid served as a control. Substrate flux through the major pathways for adenosine triphosphate generation in the heart, that is, fatty acid and glucose oxidation, was measured using [H]palmitate and [C]glucose. Biochemical analyses of nucleotides, acyl-CoAs, ceramides, and 32 acylcarnitine species were used to profile individual metabolites. Lipid rafts were isolated and used for Western blotting of the plasma membrane transporters CD36 and glucose transporter 4. RESULTS: Metabolic profiling of the hearts exposed to sevoflurane and propofol revealed distinct regulation of fatty acid and glucose oxidation. Sevoflurane selectively decreased fatty acid oxidation, which was closely related to a marked reduction in left ventricular work. In contrast, propofol at 100 microM but not 10 microM increased glucose oxidation without affecting cardiac work. Sevoflurane decreased fatty acid transporter CD36 in lipid rafts/caveolae, whereas high propofol increased pyruvate dehydrogenase activity without affecting glucose transporter 4, providing mechanisms for the fuel shifts in energy metabolism. Propofol increased ceramide formation, and Intralipid increased hydroxy acylcarnitine species. CONCLUSIONS: Anesthetics and their solvents elicit distinct metabolic profiles in the myocardium, which may have clinical implications for the already jeopardized diseased heart.
Authors:
Lianguo Wang; Kerry W S Ko; Eliana Lucchinetti; Liyan Zhang; Heinz Troxler; Martin Hersberger; Mohamed A Omar; Elena I Posse de Chaves; Gary D Lopaschuk; Alexander S Clanachan; Michael Zaugg
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Publication Detail:
Type:  Comparative Study; In Vitro; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Anesthesiology     Volume:  113     ISSN:  1528-1175     ISO Abbreviation:  Anesthesiology     Publication Date:  2010 Sep 
Date Detail:
Created Date:  2010-08-27     Completed Date:  2010-09-24     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  1300217     Medline TA:  Anesthesiology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  541-51     Citation Subset:  AIM; IM    
Affiliation:
Department of Anesthesiology and Pain Medicine, University of Alberta, Edmonton, Alberta, Canada.
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MeSH Terms
Descriptor/Qualifier:
Anesthetics, Intravenous / pharmacology
Animals
Antigens, CD36 / metabolism*
Energy Metabolism / drug effects,  physiology
Fatty Acids / metabolism*
Glucose / metabolism*
Heart / drug effects*,  physiology
Male
Metabolome / drug effects,  physiology
Methyl Ethers / pharmacology*
Myocardium / enzymology,  metabolism*
Oxidation-Reduction / drug effects
Propofol / pharmacology*
Pyruvate Dehydrogenase Complex / metabolism
Rats
Rats, Sprague-Dawley
Chemical
Reg. No./Substance:
0/Anesthetics, Intravenous; 0/Antigens, CD36; 0/Cd36 protein, rat; 0/Fatty Acids; 0/Methyl Ethers; 0/Pyruvate Dehydrogenase Complex; 2078-54-8/Propofol; 28523-86-6/sevoflurane; 50-99-7/Glucose

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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