| Membrane-linked systems preventing superoxide formation. | |
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MedLine Citation:
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PMID: 9337489 Owner: NLM Status: MEDLINE |
Abstract/OtherAbstract:
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New facts and ideas concerning the membrane-linked mechanisms preventing superoxide formation are summarised here. It is assumed that aerobic cells possess several lines of anti-ROS defence, including optimisation of the intracellular oxygen concentration, decrease in the concentration and life-time of one-electron O2 reductants such as CoQH; and mitochondrial and cell selections, i.e. elimination of mitochondria and cells producing ROS at high rate. It is postulated that ROS-dependent pore opening and ROS-dependent apoptosis are involved in mitochondrial and cell selections. |
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Authors:
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V P Skulachev |
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Publication Detail:
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Type: Journal Article; Review |
Journal Detail:
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Title: Bioscience reports Volume: 17 ISSN: 0144-8463 ISO Abbreviation: Biosci. Rep. Publication Date: 1997 Jun |
Date Detail:
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Created Date: 1997-11-24 Completed Date: 1997-11-24 Revised Date: 2005-11-16 |
Medline Journal Info:
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Nlm Unique ID: 8102797 Medline TA: Biosci Rep Country: UNITED STATES |
Other Details:
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Languages: eng Pagination: 347-66 Citation Subset: IM |
Affiliation:
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Department of Bioenergetics, A. N. Belozersky Institute of Physico-Chemical Biology, Moscow State University, Russia. |
Export Citation:
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APA/MLA Format Download EndNote Download BibTex |
| MeSH Terms | |
Descriptor/Qualifier:
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Aconitate Hydratase
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metabolism Animals Apoptosis Azotobacter / metabolism Bacteria / metabolism Cell Membrane / drug effects, metabolism* Cell Membrane Permeability DNA, Mitochondrial / drug effects, metabolism Humans Mitochondria / metabolism Oxidation-Reduction Permeability Plants / metabolism Reactive Oxygen Species / metabolism* Superoxides / metabolism* Uncoupling Agents / metabolism, pharmacology |
| Chemical | |
Reg. No./Substance:
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0/DNA, Mitochondrial; 0/Reactive Oxygen Species; 0/Uncoupling Agents; 11062-77-4/Superoxides; EC 4.2.1.3/Aconitate Hydratase |
From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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