Document Detail


Melatonin deficiency and fibrous dysplasia: might a relation exist?
MedLine Citation:
PMID:  12376077     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Fibrous dysplasia of bone might be monostotic, polystotic, or occurs as a part of McCune-Albright syndrome and Jaffe-Lichtenstein syndrome. Activating mutations of GNAS1 gene was identified in patients with fibrous dysplasia. However, fibrous dysplasia might occur in the absence of these mutations and fibrous dysplastic tissue was produced in vitro by the effects of excess exogenous cAMP on human osteogenic cells. It was proved that the fibrous dysplastic tissue is deficient in bone sialoprotein. Melatonin deficiency might be hypothesized in syndromes associated with fibrous dysplasia or formation of fibrous dysplasia-like tissue. The receptor RZR/ROR is the nuclear receptor of melatonin and the human bone sialoprotein gene contains a RZR/ROR response element. It was supposed that binding of melatonin to its membrane receptors results in changes in the levels of activity of nuclear cAMP that lead to alteration of expression of bone sialoprotein. Also, melatonin deficiency might increase cAMP in bone through its effect on prostaglandins of the E group. Further, melatonin deficiency might explain precocious puberty in cases of McCune-Albright syndrome. We might hypothesize that melatonin deficiency might play a role in development of fibrous dysplasia in some cases.
Authors:
M E Abdel-Wanis; H Tsuchiya
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Publication Detail:
Type:  Journal Article    
Journal Detail:
Title:  Medical hypotheses     Volume:  59     ISSN:  0306-9877     ISO Abbreviation:  Med. Hypotheses     Publication Date:  2002 Nov 
Date Detail:
Created Date:  2002-10-11     Completed Date:  2003-06-30     Revised Date:  2010-02-04    
Medline Journal Info:
Nlm Unique ID:  7505668     Medline TA:  Med Hypotheses     Country:  Scotland    
Other Details:
Languages:  eng     Pagination:  552-4     Citation Subset:  IM    
Affiliation:
Orthopaedic Department, Faculty of Medicine, Kanazawa University, Kanazawa, Japan. wanis307@yahoo.com
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MeSH Terms
Descriptor/Qualifier:
Bone Development / physiology
Cyclic AMP / physiology
Fibrous Dysplasia of Bone / etiology*,  genetics
GTP-Binding Protein alpha Subunits, Gs / deficiency,  genetics
Humans
Melatonin / deficiency*,  physiology
Models, Biological
Receptors, Cell Surface / genetics,  metabolism
Receptors, Cytoplasmic and Nuclear / genetics,  metabolism
Receptors, Melatonin
Second Messenger Systems / physiology
Sialoglycoproteins / genetics,  metabolism
Chemical
Reg. No./Substance:
0/Receptors, Cell Surface; 0/Receptors, Cytoplasmic and Nuclear; 0/Receptors, Melatonin; 0/Sialoglycoproteins; 0/integrin-binding sialoprotein; 60-92-4/Cyclic AMP; 73-31-4/Melatonin; EC 3.6.5.1/GTP-Binding Protein alpha Subunits, Gs

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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