Document Detail


Melanosome maturation defect in Rab38-deficient retinal pigment epithelium results in instability of immature melanosomes during transient melanogenesis.
MedLine Citation:
PMID:  17671165     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Pathways of melanosome biogenesis in retinal pigment epithelial (RPE) cells have received less attention than those of skin melanocytes. Although the bulk of melanin synthesis in RPE cells occurs embryonically, it is not clear whether adult RPE cells continue to produce melanosomes. Here, we show that progression from pmel17-positive premelanosomes to tyrosinase-positive mature melanosomes in the RPE is largely complete before birth. Loss of functional Rab38 in the "chocolate" (cht) mouse causes dramatically reduced numbers of melanosomes in adult RPE, in contrast to the mild phenotype previously shown in skin melanocytes. Choroidal melanocytes in cht mice also have reduced melanosome numbers, but a continuing low level of melanosome biogenesis gradually overcomes the defect, unlike in the RPE. Partial compensation by Rab32 that occurs in skin melanocytes is less effective in the RPE, presumably because of the short time window for melanosome biogenesis. In cht RPE, premelanosomes form but delivery of tyrosinase is impaired. Premelanosomes that fail to deposit melanin are unstable in both cht and tyrosinase-deficient RPE. Together with the high levels of cathepsin D in immature melanosomes of the RPE, our results suggest that melanin deposition may protect the maturing melanosome from the activity of lumenal acid hydrolases.
Authors:
Vanda S Lopes; Christina Wasmeier; Miguel C Seabra; Clare E Futter
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2007-08-01
Journal Detail:
Title:  Molecular biology of the cell     Volume:  18     ISSN:  1059-1524     ISO Abbreviation:  Mol. Biol. Cell     Publication Date:  2007 Oct 
Date Detail:
Created Date:  2007-09-27     Completed Date:  2007-11-27     Revised Date:  2009-11-18    
Medline Journal Info:
Nlm Unique ID:  9201390     Medline TA:  Mol Biol Cell     Country:  United States    
Other Details:
Languages:  eng     Pagination:  3914-27     Citation Subset:  IM    
Affiliation:
Molecular and Cellular Medicine, National Heart and Lung Institute, Faculty of Medicine, Imperial College London, London SW7 2AZ, United Kingdom.
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MeSH Terms
Descriptor/Qualifier:
Animals
Cathepsin D / metabolism
Cell Count
Choroid / cytology,  ultrastructure
Melanins / metabolism
Melanosomes / metabolism*,  ultrastructure
Mice
Monophenol Monooxygenase / deficiency,  metabolism
Parturition
Pigment Epithelium of Eye / enzymology,  metabolism*,  ultrastructure
Pigmentation
Protein Transport
rab GTP-Binding Proteins / deficiency*,  metabolism
Grant Support
ID/Acronym/Agency:
//Wellcome Trust
Chemical
Reg. No./Substance:
0/Melanins; EC 1.14.18.1/Monophenol Monooxygenase; EC 3.4.23.5/Cathepsin D; EC 3.6.1.-/Rab32 protein, mouse; EC 3.6.1.-/Rab38 protein, mouse; EC 3.6.1.-/rab GTP-Binding Proteins
Comments/Corrections

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