Document Detail


Melanocytes expressing MC1R polymorphisms associated with red hair color have altered MSH-ligand activated pigmentary responses in coculture with keratinocytes.
MedLine Citation:
PMID:  17960564     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The occurrence of red hair and pale skin in individuals, which is associated with UV-radiation sensitivity and increased skin cancer risk, is mainly due to polymorphisms in the melanocortin-1 receptor (MC1R) expressed in melanocytes. We have established a serum free human melanocyte-keratinocyte coculture system to study the behavior and functional abilities of melanocytes expressing MC1R red hair color (RHC) variants in order to identify differences from their wild type (WT) counterparts. This model revealed the importance of elevated calcium levels in promoting strong melanocyte interaction with the surrounding keratinocytes and resulted in a dendritic melanocyte morphology similar to that in skin. However, the dendricity response following agonist activation of the MC1R receptor by NDP-MSH peptide, was markedly enhanced in WT melanocytes in comparison to RHC strains. Analysis of mRNA expression and protein levels of the major pigmentation markers following NDP-MSH treatment distinguished the enzyme dopachrome tautomerase as preferentially upregulated in cocultures of WT strains, with negligible or a much reduced response in melanocytes with RHC variant alleles. These results highlight the use of the coculture system in determining fundamental differences in the physiology of melanocytes expressing RHC MC1R receptors and those of WT genotype, which are likely to contribute to the increased skin cancer risk for individuals that carry these variants.
Authors:
Donald W Roberts; Richard A Newton; J Helen Leonard; Richard A Sturm
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of cellular physiology     Volume:  215     ISSN:  1097-4652     ISO Abbreviation:  J. Cell. Physiol.     Publication Date:  2008 May 
Date Detail:
Created Date:  2008-02-27     Completed Date:  2008-03-25     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0050222     Medline TA:  J Cell Physiol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  344-55     Citation Subset:  IM    
Copyright Information:
(c) 2007 Wiley-Liss, Inc.
Affiliation:
Melanogenix Group, Institute for Molecular Bioscience, University of Queensland, Brisbane, Queensland, Australia.
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MeSH Terms
Descriptor/Qualifier:
Biological Markers
Calcium / metabolism
Cell Differentiation
Cells, Cultured
Coculture Techniques
Dendritic Cells / cytology
Hair Color / genetics*
Humans
Intramolecular Oxidoreductases / genetics,  metabolism
Keratinocytes / physiology*
Ligands
Melanocyte-Stimulating Hormones / metabolism*
Melanocytes / cytology,  drug effects,  metabolism,  physiology*
Osmolar Concentration
Polymorphism, Genetic*
RNA, Messenger / metabolism
Receptor, Melanocortin, Type 1 / genetics*,  metabolism*
Skin Pigmentation / physiology*
Up-Regulation
Vimentin / metabolism
alpha-MSH / analogs & derivatives,  pharmacology
Chemical
Reg. No./Substance:
0/Biological Markers; 0/Ligands; 0/RNA, Messenger; 0/Receptor, Melanocortin, Type 1; 0/Vimentin; 581-05-5/alpha-MSH; 7440-70-2/Calcium; 75921-69-6/MSH, 4-Nle-7-Phe-alpha-; 9002-79-3/Melanocyte-Stimulating Hormones; EC 5.3.-/Intramolecular Oxidoreductases; EC 5.3.3.12/dopachrome isomerase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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