| Meeting the (N-terminal) end with acetylation. | |
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MedLine Citation:
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PMID: 21854977 Owner: NLM Status: In-Data-Review |
Abstract/OtherAbstract:
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Cell-fate decisions are tightly linked to cellular energy status. In this issue, Yi et al. (2011) introduce a mechanism by which Bcl-xL lowers the threshold for apoptosis by suppressing acetyl-CoA production, which, in turn, suppresses the N-alpha-acetylation important for activation of the proapoptotic protease caspase-2. |
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Authors:
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Joshua L Andersen; Sally Kornbluth |
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Publication Detail:
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Type: Journal Article |
Journal Detail:
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Title: Cell Volume: 146 ISSN: 1097-4172 ISO Abbreviation: Cell Publication Date: 2011 Aug |
Date Detail:
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Created Date: 2011-08-22 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 0413066 Medline TA: Cell Country: United States |
Other Details:
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Languages: eng Pagination: 503-5 Citation Subset: IM |
Copyright Information:
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Copyright © 2011 Elsevier Inc. All rights reserved. |
Affiliation:
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Department of Pharmacology and Cancer Biology, Duke University Medical Center, Durham, NC 27710, USA. |
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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