Document Detail


Mechanisms of ventricular arrhythmias.
MedLine Citation:
PMID:  1728502     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Ventricular arrhythmias may result from abnormalities of impulse initiation and/or impulse propagation. The former include automatic arrhythmias, which may occur at high (normal) levels of membrane potential or at low (abnormal) levels of membrane potential. They also include triggered activity, which may result from early (occurring before complete repolarization) or delayed (occurring after complete repolarization) afterdepolarizations. Arrhythmias resulting from abnormal impulse propagation may be reentrant, determined in part by anatomic or functional conduction block, or the result of reflection. The factors determining various arrhythmogenic mechanisms are discussed.
Authors:
O Binah; M R Rosen
Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, P.H.S.; Review    
Journal Detail:
Title:  Circulation     Volume:  85     ISSN:  0009-7322     ISO Abbreviation:  Circulation     Publication Date:  1992 Jan 
Date Detail:
Created Date:  1992-02-06     Completed Date:  1992-02-06     Revised Date:  2007-11-15    
Medline Journal Info:
Nlm Unique ID:  0147763     Medline TA:  Circulation     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  I25-31     Citation Subset:  AIM; IM    
Affiliation:
Department of Physiology and Biophysics, Faculty of Medicine, The Technion, Haifa, Israel.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Arrhythmias, Cardiac / etiology*,  physiopathology
Electrophysiology
Heart / physiopathology
Heart Conduction System / physiology,  physiopathology
Heart Ventricles
Humans
Models, Cardiovascular
Reference Values
Grant Support
ID/Acronym/Agency:
HL-28223/HL/NHLBI NIH HHS; HL-28958/HL/NHLBI NIH HHS; HL-43731/HL/NHLBI NIH HHS

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Sudden cardiac death. Structure, function, and time-dependence of risk.
Next Document:  Experimental models of ventricular tachycardia and fibrillation caused by ischemia and infarction.