| Mechanisms of cardioprotection by isoflurane against I/R injury. | |
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MedLine Citation:
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PMID: 23276931 Owner: NLM Status: In-Data-Review |
Abstract/OtherAbstract:
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Myocardial infarction is responsible for most cardiovascular mortality as well as the pathogenesis of myocardial damage during and after infarction. Efforts are underway to modulate the development of ischemia-reperfusion (I-R) injury. Recently, the protective effect of isoflurane against myocardial I-R injury emerged as a possibility. Nitric oxide, nitric oxide synthases, factors related to energy metabolism, adenosine triphosphate-sensitive potassium channels, phosphatidylinositol-3-kinase and hypoxia-inducible factor1-alpha have been shown to participate in the mechanisms of cardioprotection elicited by isoflurane against I-R injury. In this review, we focus on the mechanisms of cardioprotection offered by isoflurane against I-R injury. |
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Authors:
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Xiao-E Lang; Xiong Wang; Jian-Hua Jin |
Publication Detail:
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Type: Journal Article Date: 2013-01-01 |
Journal Detail:
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Title: Frontiers in bioscience : a journal and virtual library Volume: 18 ISSN: 1093-4715 ISO Abbreviation: Front. Biosci. Publication Date: 2013 |
Date Detail:
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Created Date: 2013-01-01 Completed Date: - Revised Date: - |
Medline Journal Info:
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Nlm Unique ID: 9709506 Medline TA: Front Biosci Country: United States |
Other Details:
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Languages: eng Pagination: 387-93 Citation Subset: IM |
Affiliation:
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Department of Cardiology, First Hospital, Shanxi Medical University, Taiyuan Shanxi,030001, P. R. China. |
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From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine
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