Document Detail


Maternal-placental insulin-like growth factor (IGF) signaling and its importance to normal embryo-fetal development.
MedLine Citation:
PMID:  20803692     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
As background for an antibody-based therapeutic program against the IGF receptor, we undertook a review of available information on the early pregnancy-specific regulation and localization of IGFs, IGF-binding proteins (BPs), IGFBP-specific proteases, and the type 1 IGF receptor relative to placental maintenance, function of placental nutrient transporters, placental cellular differentiation/turnover/apoptosis, and critical hormone signaling needed to maintain pregnancy. Possible adverse outcomes of altered IGF signaling include prenatal loss, fetal growth retardation, and maldevelopment are also discussed. It appears that the IGF axes in both the conceptus and mother are important for normal embryo-fetal growth. Thus, all molecules (i.e., both small and large) that disrupt the IGF axis could be expected to have some degree of fetal consequences.
Authors:
Christopher J Bowman; Randal D Streck; Robert E Chapin
Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't; Review    
Journal Detail:
Title:  Birth defects research. Part B, Developmental and reproductive toxicology     Volume:  89     ISSN:  1542-9741     ISO Abbreviation:  Birth Defects Res. B Dev. Reprod. Toxicol.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-08-30     Completed Date:  2010-12-14     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101155115     Medline TA:  Birth Defects Res B Dev Reprod Toxicol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  339-49     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Wiley-Liss, Inc.
Affiliation:
Drug Safety Research and Development, Pfizer, Inc, Groton, Connecticut 06340, USA. christopher.j.bowman@pfizer.com
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MeSH Terms
Descriptor/Qualifier:
Animals
Embryonic Development*
Female
Fetal Development*
Fetal Growth Retardation / metabolism
Humans
Maternal-Fetal Exchange
Placenta / embryology*,  metabolism
Pregnancy
Pregnancy Complications / metabolism
Somatomedins / physiology*
Chemical
Reg. No./Substance:
0/Somatomedins

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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