Document Detail


Maternal high-fat intake predisposes nonalcoholic fatty liver disease in C57BL/6 offspring.
MedLine Citation:
PMID:  20822767     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: This work aimed to verify the hypothesis that maternal intake of high-fat diet in critical periods of pregnancy and/or suckling period predisposes nonalcoholic fatty liver disease in adult C57BL/6 mice offspring.
STUDY DESIGN: Male pups were divided into 5 groups: (1) SC, from standard chow-fed dams; (2) G, from high-fat chow (HF)-fed dams during the gestation (G) period; (3) L, from HF-fed dams during the lactation (L) period; (4) GL, from HF-fed dams during the gestation and lactation (GL) periods; and (5) GL/HF, from HF-fed dams during GL, maintaining an HF diet from postweaning to adulthood. We analyzed body mass, plasma blood, and liver structure.
RESULTS: The G offspring showed insulin resistance and lower glucose transporter-2 expression. Hepatic steatosis was present in the G, L, GL, and mainly in GL/HF offspring. Sterol regulatory element-binding protein-1c expression was higher in G, GL, and GL/HF offspring.
CONCLUSION: Programming by HF chow predisposes hepatic adverse remodeling in the liver of adult offspring.
Authors:
Bianca M Gregorio; Vanessa Souza-Mello; Jorge J Carvalho; Carlos A Mandarim-de-Lacerda; Marcia B Aguila
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Publication Detail:
Type:  Journal Article; Research Support, Non-U.S. Gov't     Date:  2010-09-06
Journal Detail:
Title:  American journal of obstetrics and gynecology     Volume:  203     ISSN:  1097-6868     ISO Abbreviation:  Am. J. Obstet. Gynecol.     Publication Date:  2010 Nov 
Date Detail:
Created Date:  2010-11-08     Completed Date:  2010-12-06     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  0370476     Medline TA:  Am J Obstet Gynecol     Country:  United States    
Other Details:
Languages:  eng     Pagination:  495.e1-8     Citation Subset:  AIM; IM    
Copyright Information:
Copyright © 2010 Mosby, Inc. All rights reserved.
Affiliation:
Institute of Biology, Laboratory of Morphometry and Cardiovascular Morphology, State University of Rio de Janeiro, Biomedical Center, Rio de Janeiro, Brazil.
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MeSH Terms
Descriptor/Qualifier:
Alanine Transaminase / metabolism
Alkaline Phosphatase / metabolism
Analysis of Variance
Animals
Blood Glucose
Blotting, Western
Body Weight
Dietary Fats / metabolism*
Disease Susceptibility / metabolism
Enzyme-Linked Immunosorbent Assay
Fatty Liver / etiology*,  metabolism
Female
Glucose Tolerance Test
Insulin / blood
Insulin Resistance
Liver / metabolism*
Male
Maternal Nutritional Physiological Phenomena / physiology*
Mice
Pregnancy
Prenatal Exposure Delayed Effects / metabolism*
Radioimmunoassay
Tumor Necrosis Factor-alpha / metabolism
Chemical
Reg. No./Substance:
0/Blood Glucose; 0/Dietary Fats; 0/Tumor Necrosis Factor-alpha; 11061-68-0/Insulin; EC 2.6.1.2/Alanine Transaminase; EC 3.1.3.1/Alkaline Phosphatase

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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