Document Detail


Maternal high-fat diet disturbs uteroplacental hemodynamics and increases the frequency of stillbirth in a nonhuman primate model of excess nutrition.
MedLine Citation:
PMID:  21447636     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Prepregnancy maternal obesity confers an increased risk of stillbirth, but the mechanisms are unknown. Maternal obesity is associated with placental inflammation. We considered that maternal diet may predispose to the increased risk of placental inflammation and stillbirth. We hypothesized that a chronic high-fat diet (HFD) is associated with abnormal uteroplacental circulation and placental inflammation. Here we used a nonhuman primate model to determine the effect of chronic HFD on the uterine and placental hemodynamics, placental histology, and inflammation in a prospective, observational study of 24 Japanese macaques. Overall, there was a statistically significant (38-56%) reduction in uterine volume blood flow from HFD animals, whether they were lean or obese. Consumption of a HFD, independent of obesity, increased placental inflammatory cytokines and the expression of Toll-like receptor 4. We show that HFD consumption by obese mothers with hyperinsulinemia also reduced volume blood flow on the fetal side of the placenta and significantly increased the frequency of both placental infarctions and stillbirth. These results suggest that a HFD, independent of obesity, decreases uterine volume blood flow. Maternal obesity and insulin resistance further exacerbates the placental dysfunction and results in an increased frequency of stillbirth.
Authors:
Antonio E Frias; Terry K Morgan; Anne E Evans; Juha Rasanen; Karen Y Oh; Kent L Thornburg; Kevin L Grove
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't     Date:  2011-03-29
Journal Detail:
Title:  Endocrinology     Volume:  152     ISSN:  1945-7170     ISO Abbreviation:  Endocrinology     Publication Date:  2011 Jun 
Date Detail:
Created Date:  2011-05-23     Completed Date:  2011-11-22     Revised Date:  2012-05-15    
Medline Journal Info:
Nlm Unique ID:  0375040     Medline TA:  Endocrinology     Country:  United States    
Other Details:
Languages:  eng     Pagination:  2456-64     Citation Subset:  AIM; IM    
Affiliation:
Department of Obstetrics and Gynecology, Oregon Health and Science University, Portland, Oregon 97239, USA. friasa@ohsu.edu
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MeSH Terms
Descriptor/Qualifier:
Animals
Cytokines / genetics,  immunology
Dietary Fats / adverse effects*,  immunology
Disease Models, Animal
Female
Hemodynamics
Humans
Macaca
Obesity / complications*,  genetics,  immunology,  physiopathology
Placenta / blood supply*,  immunology
Pregnancy
Pregnancy Complications / genetics,  immunology,  physiopathology*
Prenatal Nutritional Physiological Phenomena*
Regional Blood Flow
Stillbirth / epidemiology*
Uterus / blood supply*,  immunology
Grant Support
ID/Acronym/Agency:
K12 HD001243-10/HD/NICHD NIH HHS; K12 HD043488-06/HD/NICHD NIH HHS; P01 HD34430/HD/NICHD NIH HHS; P51 RR00163/RR/NCRR NIH HHS; R01 DK-79194/DK/NIDDK NIH HHS; R24 DK090964/DK/NIDDK NIH HHS; R24 DK090964-02/DK/NIDDK NIH HHS; R24 DK0909640-01/DK/NIDDK NIH HHS
Chemical
Reg. No./Substance:
0/Cytokines; 0/Dietary Fats

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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