Document Detail


Magnolol induces apoptosis via activation of both mitochondrial and death receptor pathways in A375-S2 cells.
MedLine Citation:
PMID:  20162409     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
Magnolol inhibited proliferation of human malignant melanoma A375-S2 cells. The drug induced oligonucleosomal fragmentation of DNA in A375-S2 cells and increased caspase-3, 8, 9 activities followed by the degradation of caspase-3 substrates, inhibitor of caspase dependent DNase (ICAD) and poly-(ADP-ribose) polymerase (PARP). Pan-caspase inhibitor (z-VADfmk), caspase-3 inhibitor (z-DEVD-fmk), capase-8 inhibitor (z-IETD-fmk), caspase-9 inhibitor (z-LEHD-fmk) and caspase-10 inhibitor (z-AEVD-fmk) inhibited magnolol-induced A375-S2 cell apoptosis. The level of anti-apoptotic mitochondrial protein Bcl-2 was up-regulated while the level of pro-apoptotic protein Bax was down-regulated. Taken together, our results indicate that magnolol induces apoptosis by activation of both mitochondrial and death receptor pathways in A375-S2 cells.
Authors:
Qingjun You; Mingqiu Li; Guoqing Jiao
Related Documents :
19147789 - Parp-14, a member of the b aggressive lymphoma family, transduces survival signals in p...
16113249 - Incomplete activation of macrophage apoptosis during intracellular replication of legio...
20494979 - Hlj1 is a novel caspase-3 substrate and its expression enhances uv-induced apoptosis in...
12048029 - Apoptosis during ectromelia orthopoxvirus infection is devdase dependent: in vitro and ...
15132839 - Oridonin induces apoptosis of hela cells via altering expression of bcl-2/bax and activ...
9707099 - Effects of poly(adp-ribose) polymerase inhibition on cell death and chromosome damage i...
2134499 - Analysis of the effects of immune cell motility and chemotaxis on target elimination dy...
18205789 - Dissecting innate immunity by germline mutagenesis.
20434119 - Changing cytokine patterns in systemic lupus: a prospective longitudinal study.
Publication Detail:
Type:  Journal Article     Date:  2010-02-17
Journal Detail:
Title:  Archives of pharmacal research     Volume:  32     ISSN:  0253-6269     ISO Abbreviation:  Arch. Pharm. Res.     Publication Date:  2009 Dec 
Date Detail:
Created Date:  2010-02-17     Completed Date:  2010-05-18     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8000036     Medline TA:  Arch Pharm Res     Country:  Korea (South)    
Other Details:
Languages:  eng     Pagination:  1789-94     Citation Subset:  IM    
Affiliation:
Department of Cardiothoracic Surgery, Wuxi The Forth Hospital, Wuxi City Huihe Road 200, 214062, Wuxi, China.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:
Antigens, CD95 / immunology
Antineoplastic Agents, Phytogenic / pharmacology*
Apoptosis / drug effects*
Biphenyl Compounds / pharmacology*
Bisbenzimidazole
Blotting, Western
Caspase 3 / metabolism
Caspase 8 / metabolism
Caspase 9 / metabolism
Cell Line, Tumor
Cell Nucleus / drug effects,  ultrastructure
Cell Survival / drug effects
Coloring Agents
DNA Fragmentation
DNA, Neoplasm / chemistry,  drug effects,  isolation & purification
Humans
Lignans / pharmacology*
Melanoma, Experimental / drug therapy,  pathology
Mitochondria / drug effects*
Proto-Oncogene Proteins c-bcl-2 / physiology
Receptors, Death Domain / agonists*
Signal Transduction / drug effects
bcl-2-Associated X Protein / physiology
Chemical
Reg. No./Substance:
0/Antigens, CD95; 0/Antineoplastic Agents, Phytogenic; 0/Biphenyl Compounds; 0/Coloring Agents; 0/DNA, Neoplasm; 0/Lignans; 0/Proto-Oncogene Proteins c-bcl-2; 0/Receptors, Death Domain; 0/bcl-2-Associated X Protein; 23491-44-3/Bisbenzimidazole; 528-43-8/magnolol; EC 3.4.22.-/Caspase 3; EC 3.4.22.-/Caspase 8; EC 3.4.22.-/Caspase 9

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Inhibitory effects of curcuminoids on passive cutaneous anaphylaxis reaction and scratching behavior...
Next Document:  Simultaneous HPLC analysis of ceramide and dihydroceramide in human hairs.