Document Detail


Magnetic resonance imaging and brain histopathology in neuropsychiatric systemic lupus erythematosus.
MedLine Citation:
PMID:  19880162     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: Magnetic resonance imaging (MRI) often demonstrates brain lesions in neuropsychiatric systemic lupus erythematosus (NPSLE). The present study compared postmortem histopathology with premortem MRI in NPSLE.
METHODS: Two hundred subjects with NPSLE were studied prospectively with MRI over a 10-year period during which 22 subjects died. In 14 subjects, a brain autopsy with histopathology, that permitted direct comparison with premortem MRI, was successfully obtained. Surface anatomy was used to determine the approximate location of individual lesions.
RESULTS: Premortem MRI findings in fatal NPSLE were small focal white matter lesions (100%), cortical atrophy (64%), ventricular dilation (57%), cerebral edema (50%), diffuse white matter abnormalities (43%), focal atrophy (36%), cerebral infarction (29%), acute leukoencephalopathy (25%), intracranial hemorrhage (21%), and calcifications (7%). Microscopic findings in fatal NPSLE included global ischemic changes (57%), parenchymal edema (50%), microhemorrhages (43%), glial hyperplasia (43%), diffuse neuronal/axonal loss (36%), resolved cerebral infarction (33%), microthomboemboli (29%), blood vessel remodeling (29%), acute cerebral infarction (14%), acute macrohemorrhages (14%), and resolved intracranial hemorrhages (7%). Cortical atrophy and ventricular dilation seen by MRI accurately predicted brain mass at autopsy (r = -0.72, P = 0.01, and r = -0.77, P = 0.01, respectively). Cerebral autopsy findings, including infarction, cerebral edema, intracranial hemorrhage, calcifications, cysts, and focal atrophy, were also predicted accurately by premortem MRI.
CONCLUSION: Brain lesions in NPSLE detected by MRI accurately represent serious underlying cerebrovascular and parenchymal brain injury on pathology.
Authors:
Wilmer L Sibbitt; William M Brooks; Mario Kornfeld; Blaine L Hart; Arthur D Bankhurst; Carlos A Roldan
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, N.I.H., Extramural     Date:  2009-10-31
Journal Detail:
Title:  Seminars in arthritis and rheumatism     Volume:  40     ISSN:  1532-866X     ISO Abbreviation:  Semin. Arthritis Rheum.     Publication Date:  2010 Aug 
Date Detail:
Created Date:  2010-08-02     Completed Date:  2010-11-09     Revised Date:  2011-02-24    
Medline Journal Info:
Nlm Unique ID:  1306053     Medline TA:  Semin Arthritis Rheum     Country:  United States    
Other Details:
Languages:  eng     Pagination:  32-52     Citation Subset:  IM    
Copyright Information:
Copyright 2010 Elsevier Inc. All rights reserved.
Affiliation:
Department of Internal Medicine, Division of Clinical Immunology and Rheumatic Diseases, University of New Mexico Heath Sciences Center, Albuquerque, NM 87131, USA. wsibbitt@salud.unm.edu
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MeSH Terms
Descriptor/Qualifier:
Adolescent
Adult
Aged
Brain / pathology*
Child
Diagnosis, Differential
Female
Follow-Up Studies
Humans
Lupus Vasculitis, Central Nervous System / diagnosis*
Magnetic Resonance Imaging / methods*
Male
Middle Aged
Prospective Studies
Reproducibility of Results
Time Factors
Young Adult
Grant Support
ID/Acronym/Agency:
R01 HL077422-01-A3/HL/NHLBI NIH HHS; R01 NS035708/NS/NINDS NIH HHS; R01 NS039123/NS/NINDS NIH HHS
Comments/Corrections
Erratum In:
Semin Arthritis Rheum. 2011 Feb;40(4):369

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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