Document Detail


MET molecular mechanisms and therapies in lung cancer.
MedLine Citation:
PMID:  20139696     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
The MET tyrosine kinase signaling pathway is upregulated in many cancers, including lung cancer. The pathway normally promotes mitosis, cell motility and cell survival; but in cancer it can also promote cell proliferation, invasion, metastasis and angiogenesis. The activating ligand, hepatocyte growth factor (HGF) is normally secreted by fibroblasts and smooth muscle cells, but can also be produced by tumor cells. MET upregulation in lung cancer is caused by overexpression and mutation. These mutations can vary with ethnicity. MET signaling affects cytoskeletal proteins such as paxillin, which participates in cell adhesion, growth and motility. Therapeutic approaches that block MET signaling are being studied, and include the use of: small interference RNA, Geldanamycin, competitive HGF homologues, decoy receptors and direct MET inhibitors such as K252a, SU11274, PHA665752 and PF2341066. It is hoped that blocking MET signaling may one day become an effective treatment for some lung cancers.
Authors:
Ryan E Lawrence; Ravi Salgia
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Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review     Date:  2010-01-16
Journal Detail:
Title:  Cell adhesion & migration     Volume:  4     ISSN:  1933-6926     ISO Abbreviation:  Cell Adh Migr     Publication Date:    2010 Jan-Mar
Date Detail:
Created Date:  2010-04-19     Completed Date:  2010-09-09     Revised Date:  2011-07-19    
Medline Journal Info:
Nlm Unique ID:  101469464     Medline TA:  Cell Adh Migr     Country:  United States    
Other Details:
Languages:  eng     Pagination:  146-52     Citation Subset:  IM    
Affiliation:
Pritzker School of Medicine, University of Chicago Medical Center, University of Chicago, Chicago, IL, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Humans
Lung Neoplasms / enzymology*,  therapy*
Proto-Oncogene Proteins c-met / chemistry,  metabolism*
Grant Support
ID/Acronym/Agency:
5P01HL058064-140009/HL/NHLBI NIH HHS; 5R01CA100750-06/CA/NCI NIH HHS; 5R01CA125541-03/CA/NCI NIH HHS
Chemical
Reg. No./Substance:
EC 2.7.10.1/Proto-Oncogene Proteins c-met
Comments/Corrections

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