Document Detail

Lung mechanics in congenital heart disease with increased and decreased pulmonary blood flow.
MedLine Citation:
PMID:  830909     Owner:  NLM     Status:  MEDLINE    
Respiratory rate, tidal volume, dynamic lung compliance, functional residual capacity, and pulmonary resistance were measured withim 24 hours of cardiac catheterization in 25 infants, 12 of whom had increased pulmonary blood flow and 13 of whom had decreased PBF. There were no differences in the two groups of patients with respect to VT and FRC. Respiratory rate and pulmonary resistance were higher in infants with increased PBF. Lung compliance was significantly lower in infants with increased PBF (4.9 ml/cm H2O) than in those with decreased PBF (8.9 ml/cm H2O) (P less than 0.01). The decrease in CL in infants with increased PBF significantly correlated with mean pulmonary artery pressure (r = 0.798). No correaltion was found between CL and left atrial pressure or magnitude of the left-to-right shunt. Compliance was normal in patients with increased PBF and normal PAP, suggesting that PAP and not PBF is the primary factor that affects CL in patients with intracardiac left-to-right shunts.
E Bancalari; M J Jesse; H Gelband; O Garcia
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Publication Detail:
Type:  Journal Article; Research Support, U.S. Gov't, Non-P.H.S.    
Journal Detail:
Title:  The Journal of pediatrics     Volume:  90     ISSN:  0022-3476     ISO Abbreviation:  J. Pediatr.     Publication Date:  1977 Feb 
Date Detail:
Created Date:  1977-02-24     Completed Date:  1977-02-24     Revised Date:  2006-11-15    
Medline Journal Info:
Nlm Unique ID:  0375410     Medline TA:  J Pediatr     Country:  UNITED STATES    
Other Details:
Languages:  eng     Pagination:  192-5     Citation Subset:  AIM; IM    
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MeSH Terms
Airway Resistance
Atrial Function
Blood Pressure
Heart Catheterization
Heart Defects, Congenital / physiopathology*
Lung / physiopathology*
Lung Compliance
Pulmonary Artery / physiology
Pulmonary Circulation*
Residual Volume
Respiratory Function Tests
Tidal Volume

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine

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