Document Detail


Low and dysregulated production of follistatin in immune cells of relapsing-remitting multiple sclerosis patients.
MedLine Citation:
PMID:  21880375     Owner:  NLM     Status:  Publisher    
Abstract/OtherAbstract:
One of the mechanisms known to play a key role in neuronal and oligodendroglial fate specification of neural stem cells (NSCs) is restriction of bone morphogenic proteins (BMP) signaling by BMP antagonists. Here, we demonstrate that follistatin mRNA and protein secreted levels in peripheral blood mononuclear cells (PBMCs) of relapsing-remitting multiple sclerosis (RR-MS) patients are significantly reduced compared to healthy controls (HC). We also observed a different profile of regulation mechanisms. Follistatin was similarly expressed and secreted by T lymphocytes and monocytes among the PBMCs of HC, and follistatin upregulation of HC was subjected to stimulation with both LPS and TNF-α. Among PBMCs of RR-MS patients, however, follistatin was found to be downregulated in their monocytes and unresponsive to stimulation with either LPS or TNF-α. Our results may shed some light on the mechanisms involved in remyelination failure in MS, which may be related to the inability of RR-MS patients' immune cells to provide a sufficient pro-neurogenic and oligodendrogenic niche, by expressing and secreting follistatin, in addition to the previously described noggin reduced expression. Our results indicate that the low expression of follistatin in immune cells of patients with RR-MS is a result of the altered immunoregulation of monocytes in these patients.
Authors:
Nataly Urshansky; Karin Mausner-Fainberg; Eitan Auriel; Keren Regev; Arnon Karni
Related Documents :
21266245 - Real-time monitoring of er stress in living cells and animals using estrap assay.
21266235 - Endoplasmic reticulum stress in brain damage.
12592565 - Immunopathogenesis of onchocerca volvulus keratitis (river blindness): a novel role for...
14688095 - Role of toll-like receptor 4 in induction of cell-mediated immunity and resistance to b...
17090655 - K1 protein of human herpesvirus 8 suppresses lymphoma cell fas-mediated apoptosis.
22094425 - Cellular homeostasis in fungi: impact on the aging process.
Publication Detail:
Type:  JOURNAL ARTICLE     Date:  2011-8-29
Journal Detail:
Title:  Journal of neuroimmunology     Volume:  -     ISSN:  1872-8421     ISO Abbreviation:  -     Publication Date:  2011 Aug 
Date Detail:
Created Date:  2011-9-1     Completed Date:  -     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  8109498     Medline TA:  J Neuroimmunol     Country:  -    
Other Details:
Languages:  ENG     Pagination:  -     Citation Subset:  -    
Copyright Information:
Copyright © 2011. Published by Elsevier B.V.
Affiliation:
Neuroimmunology Laboratory, Department of Neurology, Tel Aviv Sourasky Medical Center, Tel Aviv University, Tel Aviv, Israel.
Export Citation:
APA/MLA Format     Download EndNote     Download BibTex
MeSH Terms
Descriptor/Qualifier:

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


Previous Document:  Factors associated with presenteeism among employed Australian adults reporting lifetime major depre...
Next Document:  Foreign body reaction after cochlear implantation.