Document Detail


Low concentrations of a non-hydrolysable tetra-S-glycosylated porphyrin and low light induces apoptosis in human breast cancer cells via stress of the endoplasmic reticulum.
MedLine Citation:
PMID:  18958330     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
A water-soluble tetra-S-glycosylated porphyrin (P-Glu(4)) is absorbed by MDA-MB-231 human breast cancer cells whereupon irradiation with visible light causes necrosis or apoptosis depending on the concentration of the porphyrin and the power of the light. With the same amount of light irradiation power (9.4 W m(-2)), at 10-20 microM concentrations necrosis is predominantly observed, while at <10 microM concentrations, apoptosis is the principal cause of cell death. Of the various possible pathways for the induction of apoptosis, experiments demonstrate that calcium is released from the endoplasmic reticulum, cytochrome c is liberated from the mitochondria to the cytosol, pro-caspase-3 is activated, poly-(ADP-ribose) polymerase is cleaved, and the chromatin is condensed subsequent to photodynamic treatment of these cells. Confocal microscopy indicates a substantial portion of the P-Glu(4) is located in the endoplasmic reticulum at <10 microM. These data indicate that the photodynamic treatment of MDA-MB-231 cells using low concentrations of the P-Glu(4) porphyrin and low light induces apoptosis mostly initiated from stress produced to the endoplasmic reticulum.
Authors:
Sebastian Thompson; Xin Chen; Li Hui; Alfredo Toschi; David A Foster; Charles Michael Drain
Publication Detail:
Type:  Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.     Date:  2008-08-18
Journal Detail:
Title:  Photochemical & photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology     Volume:  7     ISSN:  1474-905X     ISO Abbreviation:  Photochem. Photobiol. Sci.     Publication Date:  2008 Nov 
Date Detail:
Created Date:  2008-10-29     Completed Date:  2009-03-12     Revised Date:  -    
Medline Journal Info:
Nlm Unique ID:  101124451     Medline TA:  Photochem Photobiol Sci     Country:  England    
Other Details:
Languages:  eng     Pagination:  1415-21     Citation Subset:  IM    
Affiliation:
Department of Chemistry and Biochemistry, Hunter College and Graduate Center of the City University of New York, New York, NY 10065, USA.
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MeSH Terms
Descriptor/Qualifier:
Animals
Apoptosis / drug effects*,  radiation effects*
Breast Neoplasms / pathology*
Calcium / metabolism
Caspases / metabolism
Cattle
Cell Line, Tumor
Cytochromes c / metabolism
Endoplasmic Reticulum / drug effects,  metabolism*,  radiation effects
Enzyme Activation / drug effects
Glycosylation
Hydrolysis
Indoles
Light*
Microscopy, Confocal
Microscopy, Fluorescence
Mitochondria / metabolism
Poly(ADP-ribose) Polymerases / metabolism
Porphyrins / chemistry*,  pharmacology*
Staining and Labeling
Stress, Physiological / drug effects,  radiation effects
Grant Support
ID/Acronym/Agency:
CA46677/CA/NCI NIH HHS; GM60654/GM/NIGMS NIH HHS; RR-03037/RR/NCRR NIH HHS
Chemical
Reg. No./Substance:
0/Indoles; 0/Porphyrins; 47165-04-8/DAPI; 7440-70-2/Calcium; 9007-43-6/Cytochromes c; EC 2.4.2.30/Poly(ADP-ribose) Polymerases; EC 3.4.22.-/Caspases

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine


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