Document Detail


Losartan-induced attenuation of blood pressure in L-NAME hypertensive rats is associated with reversal of the enhanced expression of Gi alpha proteins.
MedLine Citation:
PMID:  15106810     Owner:  NLM     Status:  MEDLINE    
Abstract/OtherAbstract:
OBJECTIVE: We have previously reported that hearts from N-[omega]-nitro-L-arginine methyl ester (L-NAME)-induced hypertensive rats exhibited an enhanced expression of Gi proteins. Since, losartan, an AT1 receptor antagonist, has been shown to attenuate the L-NAME-induced increase in blood pressure, we undertook the present studies to evaluate whether losartan-induced decreased blood pressure in this model of hypertension is associated with attenuation of enhanced expression of Gi proteins and adenylyl cyclase signalling.
METHODS: L-NAME (70 mg/kg body weight) and losartan (10 mg/kg body weight), alone or in combination, were given orally to Sprague-Dawley rats for 4 weeks. The control rats received only plain tap water. The levels of inhibitory guanine nucleotide regulatory proteins (Gi alpha-2 and Gi alpha-3) and stimulatory (Gs alpha) proteins and Gi alpha mRNA in hearts were determined by immunoblotting and Northern blotting, respectively. Adenylyl cyclase activity was determined by measuring [32P]cAMP formation from [32P]ATP.
RESULTS: Systolic blood pressure was enhanced in L-NAME-treated rats compared to control rats (164 +/- 5.2 versus 105 +/- 2 mmHg; n = 30), and was significantly attenuated by losartan treatment (164 +/- 5.2 mmHg versus 120 +/- 2.5 mmHg; n = 30). The expression of Gi alpha-2 and Gi alpha-3 proteins and their mRNA, which was enhanced in L-NAME-treated rats, was reversed by losartan treatment. However, losartan alone did not alter the levels of Gs alpha or Gi alpha proteins. In addition, the stimulatory effects of guanosine 5'-gamma-thiotriphosphate (GTPgammaS), isoproterenol, 5'-N-ethylcarboxamideadenosine (NECA), glucagon, forskolin (FSK) and sodium fluoride (NaF) on adenylyl cyclase, which were diminished in L-NAME-treated rats, were reversed by losartan treatment. Furthermore, the inhibition of forskolin-stimulated enzyme activity by low concentrations of GTPgammaS (receptor-independent Gi functions), which was significantly enhanced in L-NAME-treated rats, was attenuated by losartan treatment. In addition, losartan was able to reverse the attenuated receptor-mediated inhibitions of adenylyl cyclase by oxotremorine and angiotensin II towards control.
CONCLUSIONS: These results suggest the implication of AT1 receptors in enhanced expression of Gi alpha proteins and increased blood pressure in L-NAME-induced hypertension.
Authors:
Shehla Hashim; Madhu B Anand-Srivastava
Publication Detail:
Type:  Comparative Study; Journal Article; Research Support, Non-U.S. Gov't    
Journal Detail:
Title:  Journal of hypertension     Volume:  22     ISSN:  0263-6352     ISO Abbreviation:  J. Hypertens.     Publication Date:  2004 Jan 
Date Detail:
Created Date:  2004-04-26     Completed Date:  2004-11-06     Revised Date:  2012-07-13    
Medline Journal Info:
Nlm Unique ID:  8306882     Medline TA:  J Hypertens     Country:  England    
Other Details:
Languages:  eng     Pagination:  181-90     Citation Subset:  IM    
Affiliation:
Department of Physiology and Groupe de recherche sur le système nerveux autonome, Faculty of Medicine, University of Montreal, Montreal, Quebec, Canada.
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MeSH Terms
Descriptor/Qualifier:
Adenylate Cyclase / drug effects,  metabolism
Animals
Antihypertensive Agents / pharmacology*
Blood Pressure / drug effects*
Disease Models, Animal
Enzyme Inhibitors / pharmacology*
Forskolin / pharmacology
GTP-Binding Protein alpha Subunit, Gi2
GTP-Binding Protein alpha Subunits, Gi-Go / drug effects*,  metabolism*
Guanosine 5'-O-(3-Thiotriphosphate) / pharmacology
Hypertension / metabolism*
Losartan / pharmacology*
Male
Models, Cardiovascular
NG-Nitroarginine Methyl Ester / pharmacology*
Proto-Oncogene Proteins / drug effects*,  metabolism*
RNA, Messenger / drug effects,  metabolism
Rats
Rats, Inbred SHR
Rats, Sprague-Dawley
Sodium Fluoride / pharmacology
Statistics as Topic
Chemical
Reg. No./Substance:
0/Antihypertensive Agents; 0/Enzyme Inhibitors; 0/Proto-Oncogene Proteins; 0/RNA, Messenger; 114798-26-4/Losartan; 37589-80-3/Guanosine 5'-O-(3-Thiotriphosphate); 50903-99-6/NG-Nitroarginine Methyl Ester; 66428-89-5/Forskolin; 7681-49-4/Sodium Fluoride; EC 3.6.5.1/GTP-Binding Protein alpha Subunit, Gi2; EC 3.6.5.1/GTP-Binding Protein alpha Subunits, Gi-Go; EC 3.6.5.1/Gnai2 protein, rat; EC 4.6.1.1/Adenylate Cyclase

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